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Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes
| Autores principales: | , , , , , , , , , , , , , , , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Nature Publishing Group UK
2020
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7060176/ https://www.ncbi.nlm.nih.gov/pubmed/32296031 http://dx.doi.org/10.1038/s41392-020-0131-0 |
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| author | Wan, Yi Ching Esther Leung, Tsz Chui Sophia Ding, Dongbo Sun, Xulun Liu, Jiaxian Zhu, Lina Kang, Tze Zhen Evangeline Yang, Du Zhang, Yuchen Zhang, Jitian Qian, Chengmin Huen, Michael Shing Yan Li, Qing Chow, Maggie Zi Ying Zheng, Zongli Han, Junhong Goel, Ajay Wang, Xin Ishibashi, Toyotaka Chan, Kui Ming |
| author_facet | Wan, Yi Ching Esther Leung, Tsz Chui Sophia Ding, Dongbo Sun, Xulun Liu, Jiaxian Zhu, Lina Kang, Tze Zhen Evangeline Yang, Du Zhang, Yuchen Zhang, Jitian Qian, Chengmin Huen, Michael Shing Yan Li, Qing Chow, Maggie Zi Ying Zheng, Zongli Han, Junhong Goel, Ajay Wang, Xin Ishibashi, Toyotaka Chan, Kui Ming |
| author_sort | Wan, Yi Ching Esther |
| collection | PubMed |
| description | |
| format | Online Article Text |
| id | pubmed-7060176 |
| institution | National Center for Biotechnology Information |
| language | English |
| publishDate | 2020 |
| publisher | Nature Publishing Group UK |
| record_format | MEDLINE/PubMed |
| spelling | pubmed-70601762020-03-19 Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes Wan, Yi Ching Esther Leung, Tsz Chui Sophia Ding, Dongbo Sun, Xulun Liu, Jiaxian Zhu, Lina Kang, Tze Zhen Evangeline Yang, Du Zhang, Yuchen Zhang, Jitian Qian, Chengmin Huen, Michael Shing Yan Li, Qing Chow, Maggie Zi Ying Zheng, Zongli Han, Junhong Goel, Ajay Wang, Xin Ishibashi, Toyotaka Chan, Kui Ming Signal Transduct Target Ther Letter Nature Publishing Group UK 2020-03-06 /pmc/articles/PMC7060176/ /pubmed/32296031 http://dx.doi.org/10.1038/s41392-020-0131-0 Text en © The Author(s) 2020 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
| spellingShingle | Letter Wan, Yi Ching Esther Leung, Tsz Chui Sophia Ding, Dongbo Sun, Xulun Liu, Jiaxian Zhu, Lina Kang, Tze Zhen Evangeline Yang, Du Zhang, Yuchen Zhang, Jitian Qian, Chengmin Huen, Michael Shing Yan Li, Qing Chow, Maggie Zi Ying Zheng, Zongli Han, Junhong Goel, Ajay Wang, Xin Ishibashi, Toyotaka Chan, Kui Ming Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title | Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title_full | Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title_fullStr | Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title_full_unstemmed | Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title_short | Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes |
| title_sort | cancer-associated histone mutation h2bg53d disrupts dna–histone octamer interaction and promotes oncogenic phenotypes |
| topic | Letter |
| url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7060176/ https://www.ncbi.nlm.nih.gov/pubmed/32296031 http://dx.doi.org/10.1038/s41392-020-0131-0 |
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