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Isorhamnetin inhibited the proliferation and metastasis of androgen-independent prostate cancer cells by targeting the mitochondrion-dependent intrinsic apoptotic and PI3K/Akt/mTOR pathway

The present study investigated the effects of Isorhamnetin on two types of prostate cancer cells (androgen-independent and androgen-dependent) and explored its possible mechanisms underlying such effects. Treatment with Isorhamnetin significantly inhibited cell growth and induced lactate dehydrogena...

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Detalles Bibliográficos
Autores principales: Cai, Fangzhen, Zhang, Yanmei, Li, Jianwei, Huang, Sihuai, Gao, Ruilin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Portland Press Ltd. 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7080645/
https://www.ncbi.nlm.nih.gov/pubmed/32039440
http://dx.doi.org/10.1042/BSR20192826

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