Cargando…

Estrogen Attenuates the Growth of Human Papillomavirus-Positive Epithelial Cells

Human papillomaviruses (HPVs) are small, double-stranded DNA viruses that are significant risk factors in the development of cancer, and HPV accounts for approximately 5% of all worldwide cancers. Recent studies using data from The Cancer Genome Atlas (TCGA) have demonstrated that elevated levels of...

Descripción completa

Detalles Bibliográficos
Autores principales:	Bristol, Molly L., James, Claire D., Wang, Xu, Fontan, Christian T., Morgan, Iain M.
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	American Society for Microbiology 2020
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7082135/ https://www.ncbi.nlm.nih.gov/pubmed/32188745 http://dx.doi.org/10.1128/mSphere.00049-20

Ejemplares similares

The Relationship between Estrogen-Related Signaling and Human Papillomavirus Positive Cancers
por: James, Claire D., et al.
Publicado: (2020)

Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene Transcription
por: James, Claire D., et al.
Publicado: (2020)

Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during Mitosis
por: Prabhakar, Apurva T., et al.
Publicado: (2022)

Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer Outcomes
por: Fontan, Christian T., et al.
Publicado: (2020)

Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle
por: Prabhakar, Apurva T., et al.
Publicado: (2023)

Activating the DNA Damage Response and Suppressing Innate Immunity: Human Papillomaviruses Walk the Line
por: James, Claire D., et al.
Publicado: (2020)

An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer
por: Evans, Michael R., et al.
Publicado: (2017)

Correction: An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer
por: Evans, Michael R., et al.
Publicado: (2019)

Why Human Papillomaviruses Activate the DNA Damage Response (DDR) and How Cellular and Viral Replication Persists in the Presence of DDR Signaling
por: Bristol, Molly L., et al.
Publicado: (2017)

Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function
por: Prabhakar, Apurva T., et al.
Publicado: (2023)

Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation
por: Das, Dipon, et al.
Publicado: (2019)

Using a Human Papillomavirus Model to Study DNA Replication and Repair of Wild Type and Damaged DNA Templates in Mammalian Cells
por: Das, Dipon, et al.
Publicado: (2020)

Correction for Morgan et al., “Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation”
por: Das, Dipon, et al.
Publicado: (2019)

SAMHD1 Regulates Human Papillomavirus 16-Induced Cell Proliferation and Viral Replication during Differentiation of Keratinocytes
por: James, Claire D., et al.
Publicado: (2019)

DNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA Replication
por: Bristol, Molly L., et al.
Publicado: (2016)

A Critical Role for p53 during the HPV16 Life Cycle
por: Fontan, Christian T., et al.
Publicado: (2022)

CK2 Phosphorylation of Human Papillomavirus 16 E2 on Serine 23 Promotes Interaction with TopBP1 and Is Critical for E2 Interaction with Mitotic Chromatin and the Viral Life Cycle
por: Prabhakar, Apurva T., et al.
Publicado: (2021)

Werner Syndrome Protein (WRN) Regulates Cell Proliferation and the Human Papillomavirus 16 Life Cycle during Epithelial Differentiation
por: James, Claire D., et al.
Publicado: (2020)

Estrogen receptor α as a predictive biomarker for survival in human papillomavirus-positive oropharyngeal squamous cell carcinoma
por: Kwon, Soohyeon, et al.
Publicado: (2020)

The human papillomavirus oncoproteins: a review of the host pathways targeted on the road to transformation
por: Scarth, James A., et al.
Publicado: (2021)

Distribution and location of Daxx in cervical epithelial cells with high risk human papillomavirus positive
por: Tang, Shuang-yang, et al.
Publicado: (2014)

Human Papillomaviruses; Epithelial Tropisms, and the Development of Neoplasia
por: Egawa, Nagayasu, et al.
Publicado: (2015)

Benign epithelial oral lesions – association with human papillomavirus
por: Piña, Alicia-Rumayor, et al.
Publicado: (2019)

Human papillomavirus infection and risk of progression of epithelial abnormalities of the cervix.
por: Woodman, C. B., et al.
Publicado: (1996)

Stop! In the name of transforming growth factor-β: keeping estrogen receptor-α-positive mammary epithelial cells from proliferating
por: Grimm, Sandra L, et al.
Publicado: (2006)

Modelling estrogen receptor alpha-positive breast cancer by transformation of normal human mammary epithelial cells
por: Schmidt, X, et al.
Publicado: (2008)

Estrogen and estrogen receptors in the modulation of gastrointestinal epithelial secretion
por: Yang, Xin, et al.
Publicado: (2017)

Growth Dependence of Human Papillomavirus 16 DNA‐positive Cervical Cancer Cell Lines and Human Papillomavirus 16‐Transformed Human and Rat Cells on the Viral Oncoproteins
por: Watanabe, Sumie, et al.
Publicado: (1993)

Efficacy of EZH2 inhibitory drugs in human papillomavirus-positive and human papillomavirus-negative oropharyngeal squamous cell carcinomas
por: Lindsay, Cameron D., et al.
Publicado: (2017)

Oncogenic Human Papillomaviruses Activate the Tumor-Associated Lens Epithelial-Derived Growth Factor (LEDGF) Gene
por: Leitz, Jenny, et al.
Publicado: (2014)

Human Papillomavirus Typing in HIV-Positive Women
por: Hameed, Meera, et al.
Publicado: (2001)

Human Papillomavirus Typing in HIV-Positive Women
por: Faro, Sebastian
Publicado: (2001)

The origins of estrogen receptor alpha-positive and estrogen receptor alpha-negative human breast cancer
por: Allred, D Craig, et al.
Publicado: (2004)

Human Papillomavirus Oral- and Sero- Positivity in Fanconi Anemia
por: Sauter, Sharon L., et al.
Publicado: (2021)

Integration of Human Papillomavirus Genomes in Head and Neck Cancer: Is It Time to Consider a Paradigm Shift?
por: Morgan, Iain M., et al.
Publicado: (2017)

Epithelial-mesenchymal plasticity determines estrogen receptor positive breast cancer dormancy and epithelial reconversion drives recurrence
por: Aouad, Patrick, et al.
Publicado: (2022)

Inhibition of CDK8 mediator kinase suppresses estrogen dependent transcription and the growth of estrogen receptor positive breast cancer
por: McDermott, Martina S.J., et al.
Publicado: (2017)

Growth inhibition and growth stimulation by estradiol of estrogen receptor transfected human breast epithelial cell lines involve different pathways
por: Lundholt, BK, et al.
Publicado: (2000)

Human Papillomavirus Positive Squamous Cell Carcinoma of the Rectum
por: Makadia, Shraddhadevi, et al.
Publicado: (2020)

Human papillomavirus, gene mutation and estrogen and progesterone receptors in breast cancer: a cross-sectional study
por: Elagali, Abdallah Mohammed, et al.
Publicado: (2021)

Cannot write session to /tmp/vufind_sessions/sess_1fnvhrqbqb6f15fehr4g8cmc1h