Cargando…
Tick Saliva Protein Evasin-3 Allows for Visualization of Inflammation in Arteries through Interactions with CXC-Type Chemokines Deposited on Activated Endothelium
[Image: see text] Atherosclerosis is one of the leading causes of mortality in developed and developing countries. The onset of atherosclerosis development is accompanied by overexpression of several inflammatory chemokines. Neutralization of these chemokines by chemokine-binding agents attenuates a...
Autores principales: | Denisov, Stepan S., Heinzmann, Alexandra C. A., Vajen, Tanja, Vries, Mark H. M., Megens, Remco T. A., Suylen, Dennis, Koenen, Rory R., Post, Mark J., Ippel, Johannes H., Hackeng, Tilman M., Dijkgraaf, Ingrid |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Chemical
Society
2020
|
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7086393/ https://www.ncbi.nlm.nih.gov/pubmed/32077689 http://dx.doi.org/10.1021/acs.bioconjchem.0c00095 |
Ejemplares similares
-
Tick saliva protein Evasin-3 modulates chemotaxis by disrupting CXCL8 interactions with glycosaminoglycans and CXCR2
por: Denisov, Stepan S., et al.
Publicado: (2019) -
Structural characterization of anti-CCL5 activity of the tick salivary protein evasin-4
por: Denisov, Stepan S., et al.
Publicado: (2020) -
A knottin scaffold directs the CXC-chemokine–binding specificity of tick evasins
por: Lee, Angela W., et al.
Publicado: (2019) -
Molecular basis of anticoagulant and anticomplement activity of the tick salivary protein Salp14 and its homologs
por: Denisov, Stepan S., et al.
Publicado: (2021) -
Deletion of junctional adhesion molecule A from platelets increases early‐stage neointima formation after wire injury in hyperlipidemic mice
por: Zhao, Zhen, et al.
Publicado: (2017)