Theimunopathogenesis of cronic HBV inducedliver disease

In conclusion, until the cellular biology of hepatitis B virus infection is understood and until the technology for the development of suitable virus infected autologous target cells is available, it will not be possible to definitively establish the mechanism involved in the pathogenesis of hepatit...

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Detalles Bibliográficos
Autores principales: Levy, Gary A., Chisari, Francis V.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer-Verlag 1981
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7087516/
https://www.ncbi.nlm.nih.gov/pubmed/6168027
http://dx.doi.org/10.1007/BF01951492
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author Levy, Gary A.
Chisari, Francis V.
author_facet Levy, Gary A.
Chisari, Francis V.
author_sort Levy, Gary A.
collection PubMed
description In conclusion, until the cellular biology of hepatitis B virus infection is understood and until the technology for the development of suitable virus infected autologous target cells is available, it will not be possible to definitively establish the mechanism involved in the pathogenesis of hepatitis B virus induced hepatocellular injury. Clearly, considerable effort must now be focused on the basic biology of virus infection, replication, and effects on hepatocellular metabolism before it will be possible to perform the definitive experiments to elucidate the role played by the immune system in the pathogenesis of this disease.
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spelling pubmed-70875162020-03-23 Theimunopathogenesis of cronic HBV inducedliver disease Levy, Gary A. Chisari, Francis V. Springer Semin Immunopathol Article In conclusion, until the cellular biology of hepatitis B virus infection is understood and until the technology for the development of suitable virus infected autologous target cells is available, it will not be possible to definitively establish the mechanism involved in the pathogenesis of hepatitis B virus induced hepatocellular injury. Clearly, considerable effort must now be focused on the basic biology of virus infection, replication, and effects on hepatocellular metabolism before it will be possible to perform the definitive experiments to elucidate the role played by the immune system in the pathogenesis of this disease. Springer-Verlag 1981 /pmc/articles/PMC7087516/ /pubmed/6168027 http://dx.doi.org/10.1007/BF01951492 Text en © Springer-Verlag 1981 This article is made available via the PMC Open Access Subset for unrestricted research re-use and secondary analysis in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the World Health Organization (WHO) declaration of COVID-19 as a global pandemic.
spellingShingle Article
Levy, Gary A.
Chisari, Francis V.
Theimunopathogenesis of cronic HBV inducedliver disease
title Theimunopathogenesis of cronic HBV inducedliver disease
title_full Theimunopathogenesis of cronic HBV inducedliver disease
title_fullStr Theimunopathogenesis of cronic HBV inducedliver disease
title_full_unstemmed Theimunopathogenesis of cronic HBV inducedliver disease
title_short Theimunopathogenesis of cronic HBV inducedliver disease
title_sort theimunopathogenesis of cronic hbv inducedliver disease
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7087516/
https://www.ncbi.nlm.nih.gov/pubmed/6168027
http://dx.doi.org/10.1007/BF01951492
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