Cargando…
Fisetin Prevents HT22 Cells From High Glucose-Induced Neurotoxicity via PI3K/Akt/CREB Signaling Pathway
Hyperglycemia has been widely considered as a key risk factor for diabetic encephalopathy which can cause neuronal apoptosis and cognitive deficits. The flavonoid compound, fisetin, possesses potential neuroprotective effects and also enhances learning and memory. However, the role of fisetin in hyp...
Autores principales: | Zhang, Shenshen, Xue, Ran, Geng, Yaping, Wang, Hao, Li, Wenjie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7096699/ https://www.ncbi.nlm.nih.gov/pubmed/32265642 http://dx.doi.org/10.3389/fnins.2020.00241 |
Ejemplares similares
-
Genistein attenuates isoflurane-induced neurotoxicity and improves impaired spatial learning and memory by regulating cAMP/CREB and BDNF-TrkB-PI3K/Akt signaling
por: Jiang, Tao, et al.
Publicado: (2017) -
Fisetin inhibits the proliferation, migration and invasion of pancreatic cancer by targeting PI3K/AKT/mTOR signaling
por: Xiao, Yanyi, et al.
Publicado: (2021) -
Sevoflurane alleviates oxygen-glucose deprivation/reoxygenation-induced injury in HT22 cells through regulation of the PI3K/AKT/GSK3β signaling pathway
por: Yu, Qiong, et al.
Publicado: (2021) -
Neurotoxicity mechanism of aconitine in HT22 cells studied by microfluidic chip-mass spectrometry
por: Zhang, Yingrui, et al.
Publicado: (2023) -
Anti-cancer effects of fisetin on mammary carcinoma cells via regulation of the PI3K/Akt/mTOR pathway: In vitro and in vivo studies
por: Sun, Xu, et al.
Publicado: (2018)