A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()

Resistance of mice to mouse hepatitis virus type 3 (MHV3) infection is genetically determined. Normal adult A/J mice are resistant, and BALB/c mice are susceptible. Higher titers of virus and interferon (IFN) in vivo were found in MHV3-infected BALB/c mice compared with A/J mice. In vitro

Detalles Bibliográficos
Autores principales: Lucchiari, Maria A., Pereira, Carlos A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Gustav Fischer Verlag · Stuttgart · New York. Published by Elsevier GmbH 1989
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7134421/
https://www.ncbi.nlm.nih.gov/pubmed/2560461
http://dx.doi.org/10.1016/S0171-2985(89)80026-9
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author Lucchiari, Maria A.
Pereira, Carlos A.
author_facet Lucchiari, Maria A.
Pereira, Carlos A.
author_sort Lucchiari, Maria A.
collection PubMed
description Resistance of mice to mouse hepatitis virus type 3 (MHV3) infection is genetically determined. Normal adult A/J mice are resistant, and BALB/c mice are susceptible. Higher titers of virus and interferon (IFN) in vivo were found in MHV3-infected BALB/c mice compared with A/J mice. In vitro
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spelling pubmed-71344212020-04-08 A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance() Lucchiari, Maria A. Pereira, Carlos A. Immunobiology Article Resistance of mice to mouse hepatitis virus type 3 (MHV3) infection is genetically determined. Normal adult A/J mice are resistant, and BALB/c mice are susceptible. Higher titers of virus and interferon (IFN) in vivo were found in MHV3-infected BALB/c mice compared with A/J mice. In vitro Gustav Fischer Verlag · Stuttgart · New York. Published by Elsevier GmbH 1989-11 2011-11-02 /pmc/articles/PMC7134421/ /pubmed/2560461 http://dx.doi.org/10.1016/S0171-2985(89)80026-9 Text en © 1989 Gustav Fischer Verlag · Stuttgart · New York Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active.
spellingShingle Article
Lucchiari, Maria A.
Pereira, Carlos A.
A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title_full A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title_fullStr A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title_full_unstemmed A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title_short A Major Role of Macrophage Activation by Interferon-Gamma during Mouse Hepatitis Virus Type 3 Infection. I. Genetically Dependent Resistance()
title_sort major role of macrophage activation by interferon-gamma during mouse hepatitis virus type 3 infection. i. genetically dependent resistance()
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7134421/
https://www.ncbi.nlm.nih.gov/pubmed/2560461
http://dx.doi.org/10.1016/S0171-2985(89)80026-9
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