Cargando…
KLF4 alleviates cerebral vascular injury by ameliorating vascular endothelial inflammation and regulating tight junction protein expression following ischemic stroke
BACKGROUND: Although inflammatory cell adhesion molecules (CAMs) and anti-inflammation factor Kruppel-like transcription factor (KLF) 4 have all been reported to be induced after cerebral ischemic stroke (CIS), the close temporal and spatial relationship between expressions of CAMs and KLF4 followin...
Autores principales: | Zhang, Xinyu, Wang, Lu, Han, Zhenxiang, Dong, Jing, Pang, Defang, Fu, Yuan, Li, Longxuan |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7140364/ https://www.ncbi.nlm.nih.gov/pubmed/32264912 http://dx.doi.org/10.1186/s12974-020-01780-x |
Ejemplares similares
-
Silencing of lncRNA XIST impairs angiogenesis and exacerbates cerebral vascular injury after ischemic stroke
por: Wang, Cong, et al.
Publicado: (2021) -
The critical role of KLF4 in regulating the activation of A1/A2 reactive astrocytes following ischemic stroke
por: Wang, Cong, et al.
Publicado: (2023) -
Endothelial KLF4: Crippling Vascular Injury?
por: Tugal, Derin, et al.
Publicado: (2014) -
Tim-3 promotes tube formation and decreases tight junction formation in vascular endothelial cells
por: Cong, Yizi, et al.
Publicado: (2020) -
Phosphorylated cingulin localises GEF-H1 at tight junctions to protect vascular barriers in blood endothelial cells
por: Holzner, Silvio, et al.
Publicado: (2021)