Mycobacterium tuberculosis Heat-Shock Protein 16.3 Induces Macrophage M2 Polarization Through CCRL2/CX3CR1

Mycobacterium tuberculosis, the pathogen of tuberculosis (TB), can survive in host macrophages and induce macrophages to M2 phenotype might result in latent MTB infection. During the latent phase, the expression of MTB heat-shock protein 16.3 (Hsp16.3) is markedly increased among most of bacterial p...

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Detalles Bibliográficos
Autores principales: Zhang, Yanhao, Li, Shanshan, Liu, Qianyi, Long, Ruiying, Feng, Jihong, Qin, Huan, Li, Mao, Liu, Liping, Luo, Junmin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer US 2019
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7170987/
https://www.ncbi.nlm.nih.gov/pubmed/31748849
http://dx.doi.org/10.1007/s10753-019-01132-9

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