Cargando…
A Preliminary Study of Cu Exposure Effects upon Alzheimer’s Amyloid Pathology
A large body of evidence indicates that dysregulation of cerebral biometals (Fe, Cu, Zn) and their interactions with amyloid precursor protein (APP) and Aβ amyloid may contribute to the Alzheimer’s disease (AD) Aβ amyloid pathology. However, the molecular underpinnings associated with the interactio...
Autores principales: | Pilozzi, Alexander, Yu, Zhanyang, Carreras, Isabel, Cormier, Kerry, Hartley, Dean, Rogers, Jack, Dedeoglu, Alpaslan, Huang, Xudong |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7175127/ https://www.ncbi.nlm.nih.gov/pubmed/32155778 http://dx.doi.org/10.3390/biom10030408 |
Ejemplares similares
-
Exposure to CuO Nanoparticles Mediates NFκB Activation and Enhances Amyloid Precursor Protein Expression
por: Mou, Xiaoyang, et al.
Publicado: (2020) -
Exposure of CuO Nanoparticles Contributes to Cellular Apoptosis, Redox Stress, and Alzheimer’s Aβ Amyloidosis
por: Shi, Ying, et al.
Publicado: (2020) -
Fingolimod modulates multiple neuroinflammatory markers in a mouse model of Alzheimer’s disease
por: Aytan, Nurgul, et al.
Publicado: (2016) -
Microglial response to experimental periodontitis in a murine model of Alzheimer’s disease
por: Kantarci, Alpdogan, et al.
Publicado: (2020) -
Dual dose-dependent effects of fingolimod in a mouse model of Alzheimer’s disease
por: Carreras, Isabel, et al.
Publicado: (2019)