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α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells
Hepatocellular carcinoma (HCC) is the most common liver cancer and a major cause of adult death. The current treatments for HCC suffer from drug resistance and poor prognosis; therefore, novel therapeutic agents are urgently needed. Phytochemicals have been proposed to treat a range of cancers. Amon...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7189383/ https://www.ncbi.nlm.nih.gov/pubmed/32345994 http://dx.doi.org/10.1038/s41598-020-64004-5 |
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author | Pibiri, Monica Sulas, Pia Camboni, Tania Leoni, Vera Piera Simbula, Gabriella |
author_facet | Pibiri, Monica Sulas, Pia Camboni, Tania Leoni, Vera Piera Simbula, Gabriella |
author_sort | Pibiri, Monica |
collection | PubMed |
description | Hepatocellular carcinoma (HCC) is the most common liver cancer and a major cause of adult death. The current treatments for HCC suffer from drug resistance and poor prognosis; therefore, novel therapeutic agents are urgently needed. Phytochemicals have been proposed to treat a range of cancers. Among them, α-lipoic acid (α-LA), a naturally synthesized antioxidant found in various dietary animal and plant sources, prevents oxidant-mediated cell death in normal cells while inducing apoptosis in several cancer cell lines. Previously, we demonstrated that the treatment of hepatoma cells with α-LA induced apoptosis, which was preceded by the generation of reactive oxygen species (ROS) and activation of the p53 protein, a known inducer of mitochondria-mediated apoptosis. Several studies have shown that ROS-induced apoptosis is associated with endoplasmic reticulum (ER) stress and Unfolded Protein Response (UPR) activation. Herein, we investigated if α-LA-induced apoptosis in hepatoma cell lines was ER stress- and UPR-mediated by gene expression profiling analyses. UPR and ER stress pathways were the most up-regulated after treatment with α-LA. This finding, which has been confirmed by expression analyses of ER- and UPR-associated proteins, provides a better understanding of the molecular mechanisms behind the anti-tumoral action of α-LA on hepatoma cells. |
format | Online Article Text |
id | pubmed-7189383 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-71893832020-05-04 α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells Pibiri, Monica Sulas, Pia Camboni, Tania Leoni, Vera Piera Simbula, Gabriella Sci Rep Article Hepatocellular carcinoma (HCC) is the most common liver cancer and a major cause of adult death. The current treatments for HCC suffer from drug resistance and poor prognosis; therefore, novel therapeutic agents are urgently needed. Phytochemicals have been proposed to treat a range of cancers. Among them, α-lipoic acid (α-LA), a naturally synthesized antioxidant found in various dietary animal and plant sources, prevents oxidant-mediated cell death in normal cells while inducing apoptosis in several cancer cell lines. Previously, we demonstrated that the treatment of hepatoma cells with α-LA induced apoptosis, which was preceded by the generation of reactive oxygen species (ROS) and activation of the p53 protein, a known inducer of mitochondria-mediated apoptosis. Several studies have shown that ROS-induced apoptosis is associated with endoplasmic reticulum (ER) stress and Unfolded Protein Response (UPR) activation. Herein, we investigated if α-LA-induced apoptosis in hepatoma cell lines was ER stress- and UPR-mediated by gene expression profiling analyses. UPR and ER stress pathways were the most up-regulated after treatment with α-LA. This finding, which has been confirmed by expression analyses of ER- and UPR-associated proteins, provides a better understanding of the molecular mechanisms behind the anti-tumoral action of α-LA on hepatoma cells. Nature Publishing Group UK 2020-04-28 /pmc/articles/PMC7189383/ /pubmed/32345994 http://dx.doi.org/10.1038/s41598-020-64004-5 Text en © The Author(s) 2020 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Article Pibiri, Monica Sulas, Pia Camboni, Tania Leoni, Vera Piera Simbula, Gabriella α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title | α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title_full | α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title_fullStr | α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title_full_unstemmed | α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title_short | α-Lipoic acid induces Endoplasmic Reticulum stress-mediated apoptosis in hepatoma cells |
title_sort | α-lipoic acid induces endoplasmic reticulum stress-mediated apoptosis in hepatoma cells |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7189383/ https://www.ncbi.nlm.nih.gov/pubmed/32345994 http://dx.doi.org/10.1038/s41598-020-64004-5 |
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