Conditional deletion of HDAC4 from collagen type 2α1-expressing cells increases angiogenesis in vivo

BACKGROUND: HDAC4 is a key regulator of chondrocyte hypertrophy and skeletal development, but it is not clear whether the increase in vascular invasion at growth plates is related to HDAC4 expression. To determine it, we investigated the relationship between HDAC4 and angiogenesis in both in vivo an...

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Detalles Bibliográficos
Autores principales: Gao, Lilan, Li, Shengchun, Wei, Xiaochun, Du, Guoqing, Wei, Dennis, Wei, Lei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7193350/
https://www.ncbi.nlm.nih.gov/pubmed/32354322
http://dx.doi.org/10.1186/s10020-020-00154-6

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7193350/
https://www.ncbi.nlm.nih.gov/pubmed/32354322
http://dx.doi.org/10.1186/s10020-020-00154-6

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