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βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model

Heterotopic ossification (HO) is a poorly characterized disease with ectopic bone formation in the musculoskeletal soft tissues. HO is widely considered as a tissue repair process goes away, with endochondral ossification to be the major pathological basis. The molecular mechanism of how the residen...

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Autores principales: Zhang, Qiang, Zhang, Yan, Yan, Meijun, Zhu, Kai, Su, Qihang, Pan, Jie, Yang, Mingjie, Zhou, Dong, Tan, Jun
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7202527/
https://www.ncbi.nlm.nih.gov/pubmed/32312943
http://dx.doi.org/10.18632/aging.103060
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author Zhang, Qiang
Zhang, Yan
Yan, Meijun
Zhu, Kai
Su, Qihang
Pan, Jie
Yang, Mingjie
Zhou, Dong
Tan, Jun
author_facet Zhang, Qiang
Zhang, Yan
Yan, Meijun
Zhu, Kai
Su, Qihang
Pan, Jie
Yang, Mingjie
Zhou, Dong
Tan, Jun
author_sort Zhang, Qiang
collection PubMed
description Heterotopic ossification (HO) is a poorly characterized disease with ectopic bone formation in the musculoskeletal soft tissues. HO is widely considered as a tissue repair process goes away, with endochondral ossification to be the major pathological basis. The molecular mechanism of how the resident/recruited progenitor cells for tissue regeneration error differentiated into the chondrocytes remains unknown. Here, we found Transforming Growth Factor B Induced Gene Human Clone 3 (βig-h3) was highly expressed in the inflammation and chondrogenesis stages of a heterotopic ossification model after rat Achilles tendon injury, as well as upon chondrogenic differentiation conditions in vitro. βig-h3 functioned as an extracellular matrix protein, which was induced by TGFβ signaling, could bind to the injured tendon-derived stem cells (iTDSCs) and inhibit the attachment of iTDSCs to collagen I. Exogenous βig-h3 was also found able to accelerate the process of mesenchymal condensation of cultured iTDSCs and promote chondrogenic differentiation in vitro, and additional injection of iTDSCs could promote endochondral ossification in Achilles tendon injury model. Taken together, βig-h3 might function as an adhesion protein that inhibited the attachment of iTDSCs to collagen I (the injury site) but promoted the attachment of iTDSCs to each other, which resulted in promoting chondrogenic differentiation.
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spelling pubmed-72025272020-05-11 βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model Zhang, Qiang Zhang, Yan Yan, Meijun Zhu, Kai Su, Qihang Pan, Jie Yang, Mingjie Zhou, Dong Tan, Jun Aging (Albany NY) Research Paper Heterotopic ossification (HO) is a poorly characterized disease with ectopic bone formation in the musculoskeletal soft tissues. HO is widely considered as a tissue repair process goes away, with endochondral ossification to be the major pathological basis. The molecular mechanism of how the resident/recruited progenitor cells for tissue regeneration error differentiated into the chondrocytes remains unknown. Here, we found Transforming Growth Factor B Induced Gene Human Clone 3 (βig-h3) was highly expressed in the inflammation and chondrogenesis stages of a heterotopic ossification model after rat Achilles tendon injury, as well as upon chondrogenic differentiation conditions in vitro. βig-h3 functioned as an extracellular matrix protein, which was induced by TGFβ signaling, could bind to the injured tendon-derived stem cells (iTDSCs) and inhibit the attachment of iTDSCs to collagen I. Exogenous βig-h3 was also found able to accelerate the process of mesenchymal condensation of cultured iTDSCs and promote chondrogenic differentiation in vitro, and additional injection of iTDSCs could promote endochondral ossification in Achilles tendon injury model. Taken together, βig-h3 might function as an adhesion protein that inhibited the attachment of iTDSCs to collagen I (the injury site) but promoted the attachment of iTDSCs to each other, which resulted in promoting chondrogenic differentiation. Impact Journals 2020-04-20 /pmc/articles/PMC7202527/ /pubmed/32312943 http://dx.doi.org/10.18632/aging.103060 Text en Copyright © 2020 Zhang et al. http://creativecommons.org/licenses/by/3.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Research Paper
Zhang, Qiang
Zhang, Yan
Yan, Meijun
Zhu, Kai
Su, Qihang
Pan, Jie
Yang, Mingjie
Zhou, Dong
Tan, Jun
βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title_full βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title_fullStr βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title_full_unstemmed βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title_short βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat Achilles tendon heterotopic ossification model
title_sort βig-h3 enhances chondrogenesis via promoting mesenchymal condensation in rat achilles tendon heterotopic ossification model
topic Research Paper
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7202527/
https://www.ncbi.nlm.nih.gov/pubmed/32312943
http://dx.doi.org/10.18632/aging.103060
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