Host surface ectonucleotidase-CD73 and the opportunistic pathogen, Porphyromonas gingivalis, cross-modulation underlies a new homeostatic mechanism for chronic bacterial survival in human epithelial cells

Cell surface nucleotide-metabolizing enzyme, ectonucleotidase-CD73, has emerged as a central component of the cellular homeostatic-machinery that counterbalances the danger-molecule (extracellular-ATP)-driven proinflammatory response in immune cells. While the importance of CD73 in microbial host fi...

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Detalles Bibliográficos
Autores principales: Lee, Jaden S., Chowdhury, Nityananda, Roberts, JoAnn S., Yilmaz, Özlem
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2020
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7239027/
https://www.ncbi.nlm.nih.gov/pubmed/32419582
http://dx.doi.org/10.1080/21505594.2020.1763061

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7239027/
https://www.ncbi.nlm.nih.gov/pubmed/32419582
http://dx.doi.org/10.1080/21505594.2020.1763061

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