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Children’s Health in the Digital Age
Environmental studies, metabolic research, and state of the art research in neurobiology point towards the reduced amount of natural day and sunlight exposure of the developing child, as a consequence of increasingly long hours spent indoors online, as the single unifying source of a whole set of he...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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MDPI
2020
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7246471/ https://www.ncbi.nlm.nih.gov/pubmed/32384728 http://dx.doi.org/10.3390/ijerph17093240 |
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author | Dresp-Langley, Birgitta |
author_facet | Dresp-Langley, Birgitta |
author_sort | Dresp-Langley, Birgitta |
collection | PubMed |
description | Environmental studies, metabolic research, and state of the art research in neurobiology point towards the reduced amount of natural day and sunlight exposure of the developing child, as a consequence of increasingly long hours spent indoors online, as the single unifying source of a whole set of health risks identified worldwide, as is made clear in this review of currently available literature. Over exposure to digital environments, from abuse to addiction, now concerns even the youngest (ages 0 to 2) and triggers, as argued on the basis of clear examples herein, a chain of interdependent negative and potentially long-term metabolic changes. This leads to a deregulation of the serotonin and dopamine neurotransmitter pathways in the developing brain, currently associated with online activity abuse and/or internet addiction, and akin to that found in severe substance abuse syndromes. A general functional working model is proposed under the light of evidence brought to the forefront in this review. |
format | Online Article Text |
id | pubmed-7246471 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | MDPI |
record_format | MEDLINE/PubMed |
spelling | pubmed-72464712020-06-11 Children’s Health in the Digital Age Dresp-Langley, Birgitta Int J Environ Res Public Health Review Environmental studies, metabolic research, and state of the art research in neurobiology point towards the reduced amount of natural day and sunlight exposure of the developing child, as a consequence of increasingly long hours spent indoors online, as the single unifying source of a whole set of health risks identified worldwide, as is made clear in this review of currently available literature. Over exposure to digital environments, from abuse to addiction, now concerns even the youngest (ages 0 to 2) and triggers, as argued on the basis of clear examples herein, a chain of interdependent negative and potentially long-term metabolic changes. This leads to a deregulation of the serotonin and dopamine neurotransmitter pathways in the developing brain, currently associated with online activity abuse and/or internet addiction, and akin to that found in severe substance abuse syndromes. A general functional working model is proposed under the light of evidence brought to the forefront in this review. MDPI 2020-05-06 2020-05 /pmc/articles/PMC7246471/ /pubmed/32384728 http://dx.doi.org/10.3390/ijerph17093240 Text en © 2020 by the author. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Review Dresp-Langley, Birgitta Children’s Health in the Digital Age |
title | Children’s Health in the Digital Age |
title_full | Children’s Health in the Digital Age |
title_fullStr | Children’s Health in the Digital Age |
title_full_unstemmed | Children’s Health in the Digital Age |
title_short | Children’s Health in the Digital Age |
title_sort | children’s health in the digital age |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7246471/ https://www.ncbi.nlm.nih.gov/pubmed/32384728 http://dx.doi.org/10.3390/ijerph17093240 |
work_keys_str_mv | AT dresplangleybirgitta childrenshealthinthedigitalage |