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Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7251876/ https://www.ncbi.nlm.nih.gov/pubmed/32460807 http://dx.doi.org/10.1186/s12967-020-02375-z |
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author | Gu, Yu Huang, Fei Wang, Yanling Chen, Chaojin Wu, Shan Zhou, Shaoli Hei, Ziqing Yuan, Dongdong |
author_facet | Gu, Yu Huang, Fei Wang, Yanling Chen, Chaojin Wu, Shan Zhou, Shaoli Hei, Ziqing Yuan, Dongdong |
author_sort | Gu, Yu |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-7251876 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-72518762020-06-07 Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury Gu, Yu Huang, Fei Wang, Yanling Chen, Chaojin Wu, Shan Zhou, Shaoli Hei, Ziqing Yuan, Dongdong J Transl Med Correction BioMed Central 2020-05-27 /pmc/articles/PMC7251876/ /pubmed/32460807 http://dx.doi.org/10.1186/s12967-020-02375-z Text en © The Author(s) 2020 Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data. |
spellingShingle | Correction Gu, Yu Huang, Fei Wang, Yanling Chen, Chaojin Wu, Shan Zhou, Shaoli Hei, Ziqing Yuan, Dongdong Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title | Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title_full | Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title_fullStr | Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title_full_unstemmed | Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title_short | Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
title_sort | correction to: connexin32 plays a crucial role in ros-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury |
topic | Correction |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7251876/ https://www.ncbi.nlm.nih.gov/pubmed/32460807 http://dx.doi.org/10.1186/s12967-020-02375-z |
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