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Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism

Seizures, not a typical feature of hypoparathyroidism, may present along with myalgia and tetany in patients of hypoparathyroidism. Thorough history and examination, derangement of biochemical parameters such as hypocalcemia, hyperphosphatemia, and inappropriately low levels of parathyroid hormone,...

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Detalles Bibliográficos
Autores principales: Nageen, Tabinda, Muhammad, Syed Zaki, Jamal, Ammarah
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cureus 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7255561/
https://www.ncbi.nlm.nih.gov/pubmed/32489742
http://dx.doi.org/10.7759/cureus.7888
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author Nageen, Tabinda
Muhammad, Syed Zaki
Jamal, Ammarah
author_facet Nageen, Tabinda
Muhammad, Syed Zaki
Jamal, Ammarah
author_sort Nageen, Tabinda
collection PubMed
description Seizures, not a typical feature of hypoparathyroidism, may present along with myalgia and tetany in patients of hypoparathyroidism. Thorough history and examination, derangement of biochemical parameters such as hypocalcemia, hyperphosphatemia, and inappropriately low levels of parathyroid hormone, along with basal ganglia calcification as seen on imaging, lead to the diagnosis of hypoparathyroidism in a 10-year-old child who presented to us. Treatment with calcium and active vitamin D significantly improved his condition. In this report, we discuss the presentation and treatment of hypoparathyroidism. Early detection and follow-up at clinically appropriate intervals is important to avoid complications.
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spelling pubmed-72555612020-06-01 Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism Nageen, Tabinda Muhammad, Syed Zaki Jamal, Ammarah Cureus Endocrinology/Diabetes/Metabolism Seizures, not a typical feature of hypoparathyroidism, may present along with myalgia and tetany in patients of hypoparathyroidism. Thorough history and examination, derangement of biochemical parameters such as hypocalcemia, hyperphosphatemia, and inappropriately low levels of parathyroid hormone, along with basal ganglia calcification as seen on imaging, lead to the diagnosis of hypoparathyroidism in a 10-year-old child who presented to us. Treatment with calcium and active vitamin D significantly improved his condition. In this report, we discuss the presentation and treatment of hypoparathyroidism. Early detection and follow-up at clinically appropriate intervals is important to avoid complications. Cureus 2020-04-29 /pmc/articles/PMC7255561/ /pubmed/32489742 http://dx.doi.org/10.7759/cureus.7888 Text en Copyright © 2020, Nageen et al. http://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Endocrinology/Diabetes/Metabolism
Nageen, Tabinda
Muhammad, Syed Zaki
Jamal, Ammarah
Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title_full Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title_fullStr Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title_full_unstemmed Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title_short Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism
title_sort seizures and basal ganglia calcification caused by hypoparathyroidism
topic Endocrinology/Diabetes/Metabolism
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7255561/
https://www.ncbi.nlm.nih.gov/pubmed/32489742
http://dx.doi.org/10.7759/cureus.7888
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