Cargando…

Correction: Development of muscular dystrophy in a CRISPR-engineered mutant rabbit model with frame-disrupting ANO5 mutations

Detalles Bibliográficos
Autores principales:	Sui, Tingting, Xu, Li, Lau, Yeh Siang, Liu, Di, Liu, Tingjun, Gao, Yandi, Lai, Liangxue, Han, Renzhi, Li, Zhanjun
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Nature Publishing Group UK 2020
Materias:	Correction
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7298625/ https://www.ncbi.nlm.nih.gov/pubmed/32555216 http://dx.doi.org/10.1038/s41419-020-2659-x

Ejemplares similares

Development of muscular dystrophy in a CRISPR-engineered mutant rabbit model with frame-disrupting ANO5 mutations
por: Sui, Tingting, et al.
Publicado: (2018)

A novel rabbit model of Duchenne muscular dystrophy generated by CRISPR/Cas9
por: Sui, Tingting, et al.
Publicado: (2018)

LMNA-mutated Rabbits: A Model of Premature Aging Syndrome with Muscular Dystrophy and Dilated Cardiomyopathy
por: Sui, Tingting, et al.
Publicado: (2019)

Genetic disruption of Ano5 in mice does not recapitulate human ANO5-deficient muscular dystrophy
por: Xu, Jing, et al.
Publicado: (2015)

A novel ANO5 splicing variant in a LGMD2L patient leads to production of a truncated aggregation‐prone Ano5 peptide
por: Xu, Jing, et al.
Publicado: (2018)

Automated muscle histopathology analysis using CellProfiler
por: Lau, Yeh Siang, et al.
Publicado: (2018)

CRISPR Therapeutics for Duchenne Muscular Dystrophy
por: Erkut, Esra, et al.
Publicado: (2022)

Functional validation of the albinism-associated tyrosinase T373K SNP by CRISPR/Cas9-mediated homology-directed repair (HDR) in rabbits
por: Song, Yuning, et al.
Publicado: (2018)

CRISPR/Cas9-mediated GJA8 knockout in rabbits recapitulates human congenital cataracts
por: Yuan, Lin, et al.
Publicado: (2016)

Efficient Generation of Myostatin Gene Mutated Rabbit by CRISPR/Cas9
por: Lv, Qingyan, et al.
Publicado: (2016)

NUTRITIONAL MUSCULAR DYSTROPHY IN THE GUINEA PIG AND RABBIT
por: Goettsch, Marianne, et al.
Publicado: (1931)

CRISPR technologies for the treatment of Duchenne muscular dystrophy
por: Choi, Eunyoung, et al.
Publicado: (2021)

CRISPR/Cas9-mediated Disruption of Fibroblast Growth Factor 5 in Rabbits Results in a Systemic Long Hair Phenotype by Prolonging Anagen
por: Xu, Yuxin, et al.
Publicado: (2020)

CRISPR-Generated Animal Models of Duchenne Muscular Dystrophy
por: Lim, Kenji Rowel Q., et al.
Publicado: (2020)

Generation and Phenotype Identification of PAX4 Gene Knockout Rabbit by CRISPR/Cas9 System
por: Xu, Yuanyuan, et al.
Publicado: (2018)

α-Gal antigen-deficient rabbits with GGTA1 gene disruption via CRISPR/Cas9
por: Wei, Lina, et al.
Publicado: (2022)

CRISPR/Cas9-mediated mutation of tyrosinase (Tyr) 3′ UTR induce graying in rabbit
por: Song, Yuning, et al.
Publicado: (2017)

Cardiac lesions in Duchenne muscular dystrophy model rats with out-of-frame Dmd gene mutation mediated by CRISPR/Cas9 system
por: Miyamoto, Mao, et al.
Publicado: (2020)

Applications of CRISPR/Cas9 for the Treatment of Duchenne Muscular Dystrophy
por: Lim, Kenji Rowel Q., et al.
Publicado: (2018)

Genome-wide CRISPR screen identifies LGALS2 as an oxidative stress-responsive gene with an inhibitory function on colon tumor growth
por: Li, Haiwen, et al.
Publicado: (2020)

CRISPR-induced exon skipping is dependent on premature termination codon mutations
por: Sui, Tingting, et al.
Publicado: (2018)

Mutation of the Sp1 binding site in the 5′ flanking region of SRY causes sex reversal in rabbits
por: Song, Yuning, et al.
Publicado: (2017)

Large-scale genomic deletions mediated by CRISPR/Cas9 system
por: Song, Yuning, et al.
Publicado: (2017)

Generation of muscular dystrophy model rats with a CRISPR/Cas system
por: Nakamura, Katsuyuki, et al.
Publicado: (2014)

Targeted mutagenesis in rabbit using an engineered BhCas12b variant
por: Jia, Yingqi, et al.
Publicado: (2022)

Defective BVES-mediated feedback control of cAMP in muscular dystrophy
por: Li, Haiwen, et al.
Publicado: (2023)

Targeted Disruption of V600E-Mutant BRAF Gene by CRISPR-Cpf1
por: Yang, Meijia, et al.
Publicado: (2017)

ANO5 mutations in the Polish limb girdle muscular dystrophy patients: Effects on the protein structure
por: Jarmula, Adam, et al.
Publicado: (2019)

Novel Variants of ANO5 in Two Patients With Limb Girdle Muscular Dystrophy: Case Report
por: Katz, Matthew, et al.
Publicado: (2022)

Efficient and precise generation of Tay–Sachs disease model in rabbit by prime editing system
por: Qian, Yuqiang, et al.
Publicado: (2021)

Optogenetic modeling of human neuromuscular circuits in Duchenne muscular dystrophy with CRISPR and pharmacological corrections
por: Paredes-Redondo, Amaia, et al.
Publicado: (2021)

CRISPR-Cpf1 correction of muscular dystrophy mutations in human cardiomyocytes and mice
por: Zhang, Yu, et al.
Publicado: (2017)

Long-term Evaluation of AAV-CRISPR Genome Editing for Duchenne Muscular Dystrophy
por: Nelson, Christopher E., et al.
Publicado: (2019)

CRISPR applications for Duchenne muscular dystrophy: From animal models to potential therapies
por: Chey, Yu C. J., et al.
Publicado: (2022)

Highly efficient RNA-guided base editing in rabbit
por: Liu, Zhiquan, et al.
Publicado: (2018)

Duchenne muscular dystrophy caused by a frame-shift mutation in the acceptor splice site of intron 26
por: Meregalli, Mirella, et al.
Publicado: (2016)

Walking alone milestone combined reading-frame rule improves early prediction of Duchenne muscular dystrophy
por: Ma, Yan-li, et al.
Publicado: (2022)

BVES is a novel interactor of ANO5 and regulates myoblast differentiation
por: Li, Haiwen, et al.
Publicado: (2021)

CRISPR-Based Therapeutic Gene Editing for Duchenne Muscular Dystrophy: Advances, Challenges and Perspectives
por: Chen, Guofang, et al.
Publicado: (2022)

Empower multiplex cell and tissue-specific CRISPR-mediated gene manipulation with self-cleaving ribozymes and tRNA
por: Xu, Li, et al.
Publicado: (2017)

Cannot write session to /tmp/vufind_sessions/sess_v84ptr7kl2b2tf46j7kj0tp7gs