Cargando…
Diabetes aggravates myocardial ischaemia reperfusion injury via activating Nox2‐related programmed cell death in an AMPK‐dependent manner
Cardiovascular diseases such as myocardial ischaemia have a high fatality rate in patients with diabetes. This study was designed to expose the crosstalk between oxidative stress and AMPK, a vital molecule that controls biological energy metabolism, in myocardial ischaemia reperfusion injury (I/RI)...
Autores principales: | Wang, Chunyan, Zhu, Lijie, Yuan, Wenlin, Sun, Lingbin, Xia, Zhengyuan, Zhang, Zhongjun, Yao, Weifeng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7299688/ https://www.ncbi.nlm.nih.gov/pubmed/32351005 http://dx.doi.org/10.1111/jcmm.15318 |
Ejemplares similares
-
Interleukin‐7 aggravates myocardial ischaemia/reperfusion injury by regulating macrophage infiltration and polarization
por: Yan, Mengwen, et al.
Publicado: (2021) -
Maternal diabetes up‐regulates NOX2 and enhances myocardial ischaemia/reperfusion injury in adult offspring
por: Zhang, Lili, et al.
Publicado: (2018) -
Cardioprotective effects of melatonin against myocardial ischaemia/reperfusion injury: Activation of AMPK/Nrf2 pathway
por: Xu, Chennian, et al.
Publicado: (2021) -
Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein
por: Su, Hui, et al.
Publicado: (2013) -
Protective effects of metformin against myocardial ischemia-reperfusion injury via AMPK-dependent suppression of NOX4
por: Shi, Yan, et al.
Publicado: (2021)