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SARS-CoV-2 and the cardiovascular system
The coronavirus disease COVID-19 is a public health emergency caused by a novel coronavirus named severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2). SARS-CoV-2 infection uses the angiotensin-converting enzyme 2 (ACE2) receptor, and typically spreads through the respiratory tract. Invading...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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Elsevier B.V.
2020
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7363601/ https://www.ncbi.nlm.nih.gov/pubmed/32681935 http://dx.doi.org/10.1016/j.cca.2020.07.019 |
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author | Hafiane, Anouar |
author_facet | Hafiane, Anouar |
author_sort | Hafiane, Anouar |
collection | PubMed |
description | The coronavirus disease COVID-19 is a public health emergency caused by a novel coronavirus named severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2). SARS-CoV-2 infection uses the angiotensin-converting enzyme 2 (ACE2) receptor, and typically spreads through the respiratory tract. Invading viruses can elicit an exaggerated host immune response, frequently leading to a cytokine storm that may be fueling some COVID-19 death. This response contributes to multi-organ dysfunction. Accumulating data points to an increased cardiovascular disease morbidity, and mortality in COVID-19 patients. This brief review explores potential available evidence regarding the association between COVID-19, and cardiovascular complications. |
format | Online Article Text |
id | pubmed-7363601 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | Elsevier B.V. |
record_format | MEDLINE/PubMed |
spelling | pubmed-73636012020-07-16 SARS-CoV-2 and the cardiovascular system Hafiane, Anouar Clin Chim Acta Review The coronavirus disease COVID-19 is a public health emergency caused by a novel coronavirus named severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2). SARS-CoV-2 infection uses the angiotensin-converting enzyme 2 (ACE2) receptor, and typically spreads through the respiratory tract. Invading viruses can elicit an exaggerated host immune response, frequently leading to a cytokine storm that may be fueling some COVID-19 death. This response contributes to multi-organ dysfunction. Accumulating data points to an increased cardiovascular disease morbidity, and mortality in COVID-19 patients. This brief review explores potential available evidence regarding the association between COVID-19, and cardiovascular complications. Elsevier B.V. 2020-11 2020-07-16 /pmc/articles/PMC7363601/ /pubmed/32681935 http://dx.doi.org/10.1016/j.cca.2020.07.019 Text en © 2020 Elsevier B.V. All rights reserved. Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active. |
spellingShingle | Review Hafiane, Anouar SARS-CoV-2 and the cardiovascular system |
title | SARS-CoV-2 and the cardiovascular system |
title_full | SARS-CoV-2 and the cardiovascular system |
title_fullStr | SARS-CoV-2 and the cardiovascular system |
title_full_unstemmed | SARS-CoV-2 and the cardiovascular system |
title_short | SARS-CoV-2 and the cardiovascular system |
title_sort | sars-cov-2 and the cardiovascular system |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7363601/ https://www.ncbi.nlm.nih.gov/pubmed/32681935 http://dx.doi.org/10.1016/j.cca.2020.07.019 |
work_keys_str_mv | AT hafianeanouar sarscov2andthecardiovascularsystem |