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N-acetyl cysteine treatment preserves mitochondrial indices of functionality in the brain of hyperammonemic mice

AIM OF THE STUDY: Acute or chronic live failure could result in hyperammonemia and hepatic encephalopathy (HE). HE is a clinical complication characterized by severe cognitive dysfunction and coma. The ammonium ion (NH(4)(+)) is the most suspected toxic molecule involved in the pathogenesis of HE. N...

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Detalles Bibliográficos
Autores principales:	Mohammadi, Hamidreza, Sayad, Abolfazl, Mohammadi, Mohsen, Niknahad, Hossein, Heidari, Reza
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Termedia Publishing House 2020
Materias:	Original Paper
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7380475/ https://www.ncbi.nlm.nih.gov/pubmed/32728627 http://dx.doi.org/10.5114/ceh.2020.95814

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