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Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones
It is thought that extensional structures (extensional cracks and normal faults) generated during the post-seismic period create fluid pathways that enhance the drainage of the subducting plate interface, thus reducing the pore pressure and increasing fault strength. However, it remains to be elucid...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7398914/ https://www.ncbi.nlm.nih.gov/pubmed/32747647 http://dx.doi.org/10.1038/s41598-020-68418-z |
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author | Otsubo, Makoto Hardebeck, Jeanne L. Miyakawa, Ayumu Yamaguchi, Asuka Kimura, Gaku |
author_facet | Otsubo, Makoto Hardebeck, Jeanne L. Miyakawa, Ayumu Yamaguchi, Asuka Kimura, Gaku |
author_sort | Otsubo, Makoto |
collection | PubMed |
description | It is thought that extensional structures (extensional cracks and normal faults) generated during the post-seismic period create fluid pathways that enhance the drainage of the subducting plate interface, thus reducing the pore pressure and increasing fault strength. However, it remains to be elucidated how much pore fluid pressure decreases by the extension crack formation. Here we examined (i) the pore fluid pressure decrease, and (ii) the degree fault strength recovery by the extension crack formation during the post-seismic period by analyzing extension quartz veins exposed around the Nobeoka Thrust, southwestern Japan. The Nobeoka Trust is an on-land analog of the modern splay fault at shallow depths (~ 8 km) in the Nankai Trough. The poro-elastic model of extensional quartz vein formation indicates that the formation of extensional cracks only releases up to ~ 7–8% of the total pore fluid pressure at ~ 8 km depth. The pore pressure around the Nobeoka Thrust was close to lithostatic pressure during the entire seismic cycle. The estimated effective frictional coefficient along the Nobeoka Thrust after this small fluid-loss by the extensional crack formation does not exceed 0.15. Hence, the pore fluid pressure reduction due to the post-seismic extensional cracks contributes little to increase the fault strength of the megasplay fault. |
format | Online Article Text |
id | pubmed-7398914 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-73989142020-08-04 Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones Otsubo, Makoto Hardebeck, Jeanne L. Miyakawa, Ayumu Yamaguchi, Asuka Kimura, Gaku Sci Rep Article It is thought that extensional structures (extensional cracks and normal faults) generated during the post-seismic period create fluid pathways that enhance the drainage of the subducting plate interface, thus reducing the pore pressure and increasing fault strength. However, it remains to be elucidated how much pore fluid pressure decreases by the extension crack formation. Here we examined (i) the pore fluid pressure decrease, and (ii) the degree fault strength recovery by the extension crack formation during the post-seismic period by analyzing extension quartz veins exposed around the Nobeoka Thrust, southwestern Japan. The Nobeoka Trust is an on-land analog of the modern splay fault at shallow depths (~ 8 km) in the Nankai Trough. The poro-elastic model of extensional quartz vein formation indicates that the formation of extensional cracks only releases up to ~ 7–8% of the total pore fluid pressure at ~ 8 km depth. The pore pressure around the Nobeoka Thrust was close to lithostatic pressure during the entire seismic cycle. The estimated effective frictional coefficient along the Nobeoka Thrust after this small fluid-loss by the extensional crack formation does not exceed 0.15. Hence, the pore fluid pressure reduction due to the post-seismic extensional cracks contributes little to increase the fault strength of the megasplay fault. Nature Publishing Group UK 2020-08-03 /pmc/articles/PMC7398914/ /pubmed/32747647 http://dx.doi.org/10.1038/s41598-020-68418-z Text en © The Author(s) 2020 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Article Otsubo, Makoto Hardebeck, Jeanne L. Miyakawa, Ayumu Yamaguchi, Asuka Kimura, Gaku Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title | Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title_full | Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title_fullStr | Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title_full_unstemmed | Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title_short | Localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
title_sort | localized fluid discharge by tensile cracking during the post-seismic period in subduction zones |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7398914/ https://www.ncbi.nlm.nih.gov/pubmed/32747647 http://dx.doi.org/10.1038/s41598-020-68418-z |
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