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Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction

PURPOSE: Risk factors of negative affect, body dissatisfaction, distress tolerance, and negative urgency are independently associated with bulimia symptoms and non-suicidal self-injury (NSSI). However, relationships of these risk factors within comorbid presentations are not fully understood. The cu...

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Autores principales: Hovrud, Lindsey, Simons, Raluca, Simons, Jeffrey, Korkow, John
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer International Publishing 2019
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7399667/
https://www.ncbi.nlm.nih.gov/pubmed/31292855
http://dx.doi.org/10.1007/s40519-019-00741-5
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author Hovrud, Lindsey
Simons, Raluca
Simons, Jeffrey
Korkow, John
author_facet Hovrud, Lindsey
Simons, Raluca
Simons, Jeffrey
Korkow, John
author_sort Hovrud, Lindsey
collection PubMed
description PURPOSE: Risk factors of negative affect, body dissatisfaction, distress tolerance, and negative urgency are independently associated with bulimia symptoms and non-suicidal self-injury (NSSI). However, relationships of these risk factors within comorbid presentations are not fully understood. The current study examined specific roles of these risk factors within this relationship. METHODS: An at-risk community sample of young adults (N = 429) completed an online survey of negative affect, body dissatisfaction, distress tolerance, negative urgency, bulimia symptoms, and NSSI. RESULTS: A hypothesized path model was a good fit to the data. Results indicated direct paths from body dissatisfaction, negative urgency, and distress tolerance to bulimia symptoms. Negative urgency, distress tolerance, and bulimia symptoms were directly associated with NSSI. Consistent with hypotheses, distress tolerance and negative urgency acted as vulnerability factors, increasing the strength of associations between bulimia symptoms and NSSI. Distress tolerance also strengthened associations between negative urgency and NSSI. In addition to the direct effect, negative urgency was indirectly associated with NSSI via body dissatisfaction bulimia. Hypothesized indirect effects through distress tolerance were not supported. CONCLUSIONS: Results support etiological models of bulimia and NSSI, and suggest deficits in emotion regulation strengthen risk of comorbid presentations. Furthermore, individuals with greater impulsivity and difficulty tolerating distress are at increased risk of engaging in both bulimia behaviors and NSSI, providing targets for clinical intervention. LEVEL OF EVIDENCE: Level V, cross-sectional descriptive study.
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spelling pubmed-73996672020-08-13 Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction Hovrud, Lindsey Simons, Raluca Simons, Jeffrey Korkow, John Eat Weight Disord Original Article PURPOSE: Risk factors of negative affect, body dissatisfaction, distress tolerance, and negative urgency are independently associated with bulimia symptoms and non-suicidal self-injury (NSSI). However, relationships of these risk factors within comorbid presentations are not fully understood. The current study examined specific roles of these risk factors within this relationship. METHODS: An at-risk community sample of young adults (N = 429) completed an online survey of negative affect, body dissatisfaction, distress tolerance, negative urgency, bulimia symptoms, and NSSI. RESULTS: A hypothesized path model was a good fit to the data. Results indicated direct paths from body dissatisfaction, negative urgency, and distress tolerance to bulimia symptoms. Negative urgency, distress tolerance, and bulimia symptoms were directly associated with NSSI. Consistent with hypotheses, distress tolerance and negative urgency acted as vulnerability factors, increasing the strength of associations between bulimia symptoms and NSSI. Distress tolerance also strengthened associations between negative urgency and NSSI. In addition to the direct effect, negative urgency was indirectly associated with NSSI via body dissatisfaction bulimia. Hypothesized indirect effects through distress tolerance were not supported. CONCLUSIONS: Results support etiological models of bulimia and NSSI, and suggest deficits in emotion regulation strengthen risk of comorbid presentations. Furthermore, individuals with greater impulsivity and difficulty tolerating distress are at increased risk of engaging in both bulimia behaviors and NSSI, providing targets for clinical intervention. LEVEL OF EVIDENCE: Level V, cross-sectional descriptive study. Springer International Publishing 2019-07-10 2020 /pmc/articles/PMC7399667/ /pubmed/31292855 http://dx.doi.org/10.1007/s40519-019-00741-5 Text en © The Author(s) 2019 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
spellingShingle Original Article
Hovrud, Lindsey
Simons, Raluca
Simons, Jeffrey
Korkow, John
Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title_full Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title_fullStr Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title_full_unstemmed Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title_short Non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
title_sort non-suicidal self-injury and bulimia: the role of emotion dysregulation and body dissatisfaction
topic Original Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7399667/
https://www.ncbi.nlm.nih.gov/pubmed/31292855
http://dx.doi.org/10.1007/s40519-019-00741-5
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