Testicular blood supply is altered in the 41,XX(Y)* Klinefelter syndrome mouse model

Hypergonadotropic hypogonadism is a major feature of Klinefelter syndrome (KS), assumed to be caused by testicular hormone resistance. It was previously shown that intratesticular testosterone levels in vivo and Leydig cell function in vitro seem to be normal indicating other functional constraints....

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Detalles Bibliográficos
Autores principales: Wistuba, Joachim, Beumer, Cristin, Warmeling, Ann-Sophie, Sandhowe-Klaverkamp, Reinhild, Stypmann, Jörg, Kuhlmann, Michael, Holtmeier, Richard, Damm, Oliver S., Tüttelmann, Frank, Gromoll, Jörg
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7462989/
https://www.ncbi.nlm.nih.gov/pubmed/32873847
http://dx.doi.org/10.1038/s41598-020-71377-0

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7462989/
https://www.ncbi.nlm.nih.gov/pubmed/32873847
http://dx.doi.org/10.1038/s41598-020-71377-0

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