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Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis

AIM: To highlight the occurrence of cardiac arrest due to hyperkalemia in diabetic ketoacidosis (DKA). BACKGROUND: Diabetic ketoacidosis is a commonly encountered condition. These patients can have normal or mildly elevated levels of potassium. Our patient had severe hyperkalemia due to DKA resultin...

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Autores principales: Manappallil, Robin G, Nambiar, Jayasree
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Jaypee Brothers Medical Publishers 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7519603/
https://www.ncbi.nlm.nih.gov/pubmed/33024389
http://dx.doi.org/10.5005/jp-journals-10071-23526
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author Manappallil, Robin G
Nambiar, Jayasree
author_facet Manappallil, Robin G
Nambiar, Jayasree
author_sort Manappallil, Robin G
collection PubMed
description AIM: To highlight the occurrence of cardiac arrest due to hyperkalemia in diabetic ketoacidosis (DKA). BACKGROUND: Diabetic ketoacidosis is a commonly encountered condition. These patients can have normal or mildly elevated levels of potassium. Our patient had severe hyperkalemia due to DKA resulting in cardiac arrest. Her high potassium diet and use of angiotensin receptor blocker along with acute kidney injury (AKI) would have also contributed to hyperkalemia. CASE DESCRIPTION: A 58-year-old female, known case of diabetes mellitus on insulin therapy and hypertension on telmisartan, presented with nausea, vomiting, and abdominal pain. She was diagnosed to have DKA with AKI precipitated by missed insulin and urinary tract infection. She was also on high potassium diet. Her electrocardiogram showed sinus bradycardia with prolonged QRS interval. Her potassium levels were elevated. She soon went into asystole and cardiac arrest and was resuscitated. Diabetic ketoacidosis protocols were followed along with antibiotics, and the patient improved. CONCLUSION: Severe hyperkalemia in DKA is uncommon, and this hyperkalemia resulting in cardiac arrest is an unreported scenario. Potassium correction along with DKA management protocol forms the mainstay of treatment. CLINICAL SIGNIFICANCE: Mild to moderate elevation in serum potassium occurs frequently in DKA. However, severe hyperkalemia is uncommon and is likely to be the result of insulin deficiency, acidosis, hyperosmolality, severe dehydration, and renal potassium retention. Such elevated level of potassium requires urgent correction in order to prevent cardiac arrest. HOW TO CITE THIS ARTICLE: Manappallil RG, Nambiar J. Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis. Indian J Crit Care Med 2020;24(8):737–738.
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spelling pubmed-75196032020-10-05 Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis Manappallil, Robin G Nambiar, Jayasree Indian J Crit Care Med Letter to the Editor AIM: To highlight the occurrence of cardiac arrest due to hyperkalemia in diabetic ketoacidosis (DKA). BACKGROUND: Diabetic ketoacidosis is a commonly encountered condition. These patients can have normal or mildly elevated levels of potassium. Our patient had severe hyperkalemia due to DKA resulting in cardiac arrest. Her high potassium diet and use of angiotensin receptor blocker along with acute kidney injury (AKI) would have also contributed to hyperkalemia. CASE DESCRIPTION: A 58-year-old female, known case of diabetes mellitus on insulin therapy and hypertension on telmisartan, presented with nausea, vomiting, and abdominal pain. She was diagnosed to have DKA with AKI precipitated by missed insulin and urinary tract infection. She was also on high potassium diet. Her electrocardiogram showed sinus bradycardia with prolonged QRS interval. Her potassium levels were elevated. She soon went into asystole and cardiac arrest and was resuscitated. Diabetic ketoacidosis protocols were followed along with antibiotics, and the patient improved. CONCLUSION: Severe hyperkalemia in DKA is uncommon, and this hyperkalemia resulting in cardiac arrest is an unreported scenario. Potassium correction along with DKA management protocol forms the mainstay of treatment. CLINICAL SIGNIFICANCE: Mild to moderate elevation in serum potassium occurs frequently in DKA. However, severe hyperkalemia is uncommon and is likely to be the result of insulin deficiency, acidosis, hyperosmolality, severe dehydration, and renal potassium retention. Such elevated level of potassium requires urgent correction in order to prevent cardiac arrest. HOW TO CITE THIS ARTICLE: Manappallil RG, Nambiar J. Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis. Indian J Crit Care Med 2020;24(8):737–738. Jaypee Brothers Medical Publishers 2020-08 /pmc/articles/PMC7519603/ /pubmed/33024389 http://dx.doi.org/10.5005/jp-journals-10071-23526 Text en Copyright © 2020; Jaypee Brothers Medical Publishers (P) Ltd. © The Author(s). 2020 Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (https://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted use, distribution, and non-commercial reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
spellingShingle Letter to the Editor
Manappallil, Robin G
Nambiar, Jayasree
Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title_full Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title_fullStr Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title_full_unstemmed Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title_short Hyperkalemic Cardiac Arrest in a Patient with Diabetic Ketoacidosis
title_sort hyperkalemic cardiac arrest in a patient with diabetic ketoacidosis
topic Letter to the Editor
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7519603/
https://www.ncbi.nlm.nih.gov/pubmed/33024389
http://dx.doi.org/10.5005/jp-journals-10071-23526
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