Interference with pancreatic sympathetic signaling halts the onset of diabetes in mice

The notably lobular distribution of immune lesions in type 1 diabetes (T1D) has been hypothesized to be the result of innervation within the pancreas. To investigate whether neuroimmune interactions could explain this phenomenon, we explored the impact of sympathetic signaling in the RIP-LCMV-GP mou...

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Detalles Bibliográficos
Autores principales: Christoffersson, Gustaf, Ratliff, Sowbarnika S., von Herrath, Matthias G.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Association for the Advancement of Science 2020
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7531904/
https://www.ncbi.nlm.nih.gov/pubmed/33052874
http://dx.doi.org/10.1126/sciadv.abb2878

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7531904/
https://www.ncbi.nlm.nih.gov/pubmed/33052874
http://dx.doi.org/10.1126/sciadv.abb2878

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