DEFA1B inhibits ZIKV replication and retards cell cycle progression through interaction with ORC1

AIMS: Zika virus (ZIKV) infection causes a public health concern because of its potential association with the development of microcephaly. During viral infections, the host innate immune response is mounted quickly to produce some endogenous functional molecules to limit virus replication and sprea...

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Detalles Bibliográficos
Autores principales: Li, Shuang, Zhu, Anjing, Ren, Kai, Li, Shilin, Chen, Limin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier Inc. 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7567675/
https://www.ncbi.nlm.nih.gov/pubmed/33075374
http://dx.doi.org/10.1016/j.lfs.2020.118564

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7567675/
https://www.ncbi.nlm.nih.gov/pubmed/33075374
http://dx.doi.org/10.1016/j.lfs.2020.118564

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