CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?

Detalles Bibliográficos
Autores principales: Stölting, Gabriel, Scholl, Ute I
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2020
Materias:
Commentary
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7609131/
https://www.ncbi.nlm.nih.gov/pubmed/33142317
http://dx.doi.org/10.1210/endocr/bqaa173
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author Stölting, Gabriel
Scholl, Ute I
author_facet Stölting, Gabriel
Scholl, Ute I
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spelling pubmed-76091312020-11-09 CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit? Stölting, Gabriel Scholl, Ute I Endocrinology Commentary Oxford University Press 2020-11-03 /pmc/articles/PMC7609131/ /pubmed/33142317 http://dx.doi.org/10.1210/endocr/bqaa173 Text en © The Author(s) 2020. Published by Oxford University Press on behalf of the Endocrine Society. https://creativecommons.org/licenses/by/4.0/This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) ), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Commentary
Stölting, Gabriel
Scholl, Ute I
CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title_full CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title_fullStr CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title_full_unstemmed CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title_short CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?
title_sort cacna1h calcium channel mutations in primary aldosteronism – is sodium the culprit?
topic Commentary
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7609131/
https://www.ncbi.nlm.nih.gov/pubmed/33142317
http://dx.doi.org/10.1210/endocr/bqaa173
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