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The Bezold–Jarisch reflex in a patient with coronary spastic angina

Acute inferior myocardial damage can induce transient bradycardia and hypotension—the Bezold–Jarisch reflex, which is explained by the preferential distribution of vagal nerves in the inferior wall of the left ventricle. We report a 76‐year‐old man who showed a perfusion defect in the inferior wall...

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Autores principales: Sakai, Chieko, Kawasaki, Tatsuya, Sugihara, Hiroki, Matoba, Satoaki
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7679827/
https://www.ncbi.nlm.nih.gov/pubmed/32335982
http://dx.doi.org/10.1111/anec.12759
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author Sakai, Chieko
Kawasaki, Tatsuya
Sugihara, Hiroki
Matoba, Satoaki
author_facet Sakai, Chieko
Kawasaki, Tatsuya
Sugihara, Hiroki
Matoba, Satoaki
author_sort Sakai, Chieko
collection PubMed
description Acute inferior myocardial damage can induce transient bradycardia and hypotension—the Bezold–Jarisch reflex, which is explained by the preferential distribution of vagal nerves in the inferior wall of the left ventricle. We report a 76‐year‐old man who showed a perfusion defect in the inferior wall with redistribution on exercise scintigraphy with thallium‐201. Of note, during exercise at an intensity of 100 watts, the patient's heart rate transiently decreased from 122 to 95 bpm in sinus rhythm, accompanied by ST‐segment depression. A diagnosis of coronary spastic angina was made since no stenotic lesions were observed on conventional coronary angiography.
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spelling pubmed-76798272020-11-27 The Bezold–Jarisch reflex in a patient with coronary spastic angina Sakai, Chieko Kawasaki, Tatsuya Sugihara, Hiroki Matoba, Satoaki Ann Noninvasive Electrocardiol Case Reports Acute inferior myocardial damage can induce transient bradycardia and hypotension—the Bezold–Jarisch reflex, which is explained by the preferential distribution of vagal nerves in the inferior wall of the left ventricle. We report a 76‐year‐old man who showed a perfusion defect in the inferior wall with redistribution on exercise scintigraphy with thallium‐201. Of note, during exercise at an intensity of 100 watts, the patient's heart rate transiently decreased from 122 to 95 bpm in sinus rhythm, accompanied by ST‐segment depression. A diagnosis of coronary spastic angina was made since no stenotic lesions were observed on conventional coronary angiography. John Wiley and Sons Inc. 2020-04-26 /pmc/articles/PMC7679827/ /pubmed/32335982 http://dx.doi.org/10.1111/anec.12759 Text en © 2020 The Authors. Annals of Noninvasive Electrocardiology published by Wiley Periodicals, Inc. This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.
spellingShingle Case Reports
Sakai, Chieko
Kawasaki, Tatsuya
Sugihara, Hiroki
Matoba, Satoaki
The Bezold–Jarisch reflex in a patient with coronary spastic angina
title The Bezold–Jarisch reflex in a patient with coronary spastic angina
title_full The Bezold–Jarisch reflex in a patient with coronary spastic angina
title_fullStr The Bezold–Jarisch reflex in a patient with coronary spastic angina
title_full_unstemmed The Bezold–Jarisch reflex in a patient with coronary spastic angina
title_short The Bezold–Jarisch reflex in a patient with coronary spastic angina
title_sort bezold–jarisch reflex in a patient with coronary spastic angina
topic Case Reports
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7679827/
https://www.ncbi.nlm.nih.gov/pubmed/32335982
http://dx.doi.org/10.1111/anec.12759
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