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Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression
Accumulating studies suggest that circular RNAs (circRNAs) function as key regulators in human cancers. We found that hsa_circ_0001869 participated in non-small cell lung cancer (NSCLC) progression. However, its expression and function during NSCLC remain unknown. The data advised that hsa_circ_0001...
Autores principales: | , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7762492/ https://www.ncbi.nlm.nih.gov/pubmed/33221767 http://dx.doi.org/10.18632/aging.104037 |
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author | Xu, Pei Wang, Lei Xie, Xiao Hu, Fengqing Yang, Qi Hu, Rui Jiang, Lianyong Ding, Fangbao Mei, Ju Liu, Jianhui Xiao, Haibo |
author_facet | Xu, Pei Wang, Lei Xie, Xiao Hu, Fengqing Yang, Qi Hu, Rui Jiang, Lianyong Ding, Fangbao Mei, Ju Liu, Jianhui Xiao, Haibo |
author_sort | Xu, Pei |
collection | PubMed |
description | Accumulating studies suggest that circular RNAs (circRNAs) function as key regulators in human cancers. We found that hsa_circ_0001869 participated in non-small cell lung cancer (NSCLC) progression. However, its expression and function during NSCLC remain unknown. The data advised that hsa_circ_0001869 expression was increased in NSCLC cell lines and tissues. High hsa_circ_0001869 expression had negatively correlation with the NSCLC patients prognosis. Bioinformatics and luciferase report analyses confirmed that miR-638 and FOSL2 were hsa_circ_0001869 downstream target. hsa_circ_0001869 downregulation decreased tumor proliferation, invasion and migration by promoting miR-638 expression and decreasing FOSL2 expression. As a result of overexpression of FOSL2 or silencing of miR-638, the recovery of proliferation, migration, and invasion after hsa_circ_0001869 silencing. Overexpression of FOSL2 also led to recovery of migration, invasion and proliferation after upregulation of miR-638. In vivo studies confirmed that overexpression of FOSL2 or silencing of miR-638 led to the recovery of tumor growth ability regarding A549 cells after hsa_circ_0001869 knockdown. Present investigation discovered that hsa_circ_0001869 enhanced NSCLC progression via sponging miR-638 and promoting FOSL2 expression. hsa_circ_0001869 downregulation suppressed tumor growth and invasion ability. |
format | Online Article Text |
id | pubmed-7762492 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | Impact Journals |
record_format | MEDLINE/PubMed |
spelling | pubmed-77624922021-01-08 Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression Xu, Pei Wang, Lei Xie, Xiao Hu, Fengqing Yang, Qi Hu, Rui Jiang, Lianyong Ding, Fangbao Mei, Ju Liu, Jianhui Xiao, Haibo Aging (Albany NY) Research Paper Accumulating studies suggest that circular RNAs (circRNAs) function as key regulators in human cancers. We found that hsa_circ_0001869 participated in non-small cell lung cancer (NSCLC) progression. However, its expression and function during NSCLC remain unknown. The data advised that hsa_circ_0001869 expression was increased in NSCLC cell lines and tissues. High hsa_circ_0001869 expression had negatively correlation with the NSCLC patients prognosis. Bioinformatics and luciferase report analyses confirmed that miR-638 and FOSL2 were hsa_circ_0001869 downstream target. hsa_circ_0001869 downregulation decreased tumor proliferation, invasion and migration by promoting miR-638 expression and decreasing FOSL2 expression. As a result of overexpression of FOSL2 or silencing of miR-638, the recovery of proliferation, migration, and invasion after hsa_circ_0001869 silencing. Overexpression of FOSL2 also led to recovery of migration, invasion and proliferation after upregulation of miR-638. In vivo studies confirmed that overexpression of FOSL2 or silencing of miR-638 led to the recovery of tumor growth ability regarding A549 cells after hsa_circ_0001869 knockdown. Present investigation discovered that hsa_circ_0001869 enhanced NSCLC progression via sponging miR-638 and promoting FOSL2 expression. hsa_circ_0001869 downregulation suppressed tumor growth and invasion ability. Impact Journals 2020-11-20 /pmc/articles/PMC7762492/ /pubmed/33221767 http://dx.doi.org/10.18632/aging.104037 Text en Copyright © 2020 Xu et al. https://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/3.0/) (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. |
spellingShingle | Research Paper Xu, Pei Wang, Lei Xie, Xiao Hu, Fengqing Yang, Qi Hu, Rui Jiang, Lianyong Ding, Fangbao Mei, Ju Liu, Jianhui Xiao, Haibo Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title | Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title_full | Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title_fullStr | Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title_full_unstemmed | Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title_short | Hsa_circ_0001869 promotes NSCLC progression via sponging miR-638 and enhancing FOSL2 expression |
title_sort | hsa_circ_0001869 promotes nsclc progression via sponging mir-638 and enhancing fosl2 expression |
topic | Research Paper |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7762492/ https://www.ncbi.nlm.nih.gov/pubmed/33221767 http://dx.doi.org/10.18632/aging.104037 |
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