Cargando…
ATPIF1 maintains normal mitochondrial structure which is impaired by CCM3 deficiency in endothelial cells
BACKGROUND: Numerous signaling pathways have been demonstrated experimentally to affect the pathogenesis of cerebral cavernous malformations (CCM), a disease that can be caused by CCM3 deficiency. However, the understanding of the CCM progression is still limited. The objective of the present work w...
Autores principales: | Wang, Kang, Chen, Haixuan, Zhou, Zhongyang, Zhang, Haifeng, Zhou, Huanjiao Jenny, Min, Wang |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7796565/ https://www.ncbi.nlm.nih.gov/pubmed/33422124 http://dx.doi.org/10.1186/s13578-020-00514-z |
Ejemplares similares
-
Mitochondrial Atpif1 regulates heme synthesis in developing erythroblasts
por: Shah, Dhvanit I., et al.
Publicado: (2012) -
CCM3 and cerebral cavernous malformation disease
por: Wang, Kang, et al.
Publicado: (2019) -
Caveolae-mediated Tie2 signaling contributes to CCM pathogenesis in a brain endothelial cell-specific Pdcd10-deficient mouse model
por: Zhou, Huanjiao Jenny, et al.
Publicado: (2021) -
Augmented endothelial exocytosis of angiopoietin-2 resulting from CCM3-deficiency contributes to the progression of cerebral cavernous malformation
por: Jenny Zhou, Huanjiao, et al.
Publicado: (2016) -
Upregulation of mitochondrial ATPase inhibitory factor 1 (ATPIF1) mediates increased glycolysis in mouse hearts
por: Zhou, Bo, et al.
Publicado: (2022)