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Sesn2 attenuates the damage of endothelial progenitor cells induced by angiotensin II through regulating the Keap1/Nrf2 signal pathway

Endothelial progenitor cell (EPC) dysfunction is an important physiopathological mechanism in the dynamics of the formation of atherosclerosis. It has been reported that angiotensin II (Ang-II) damages the function of EPCs in atherosclerotic plaque through induction of oxidative stress. Sestrin 2 (S...

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Detalles Bibliográficos
Autores principales:	Ding, Shiao, Ma, Nan, Liu, Hao, Tang, Min, Mei, Ju
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Impact Journals 2020
Materias:	Research Paper
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7803511/ https://www.ncbi.nlm.nih.gov/pubmed/33231566 http://dx.doi.org/10.18632/aging.104156

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