Nicorandil inhibits cardiomyocyte apoptosis and improves cardiac function by suppressing the HtrA2/XIAP/PARP signaling after coronary microembolization in rats

Cardiomyocyte apoptosis is a key factor in the deterioration of cardiac function after coronary microembolization (CME). Nicorandil (NIC) affects myocardial injury, which may be related to the inhibition of apoptosis. However, the specific mechanism of cardioprotection has not been elucidated. There...

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Detalles Bibliográficos
Autores principales: Zheng, Jing, Long, Manyun, Qin, Zhenbai, Wang, Fen, Chen, Zhiqing, Li, Lang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7809785/
https://www.ncbi.nlm.nih.gov/pubmed/33448699
http://dx.doi.org/10.1002/prp2.699

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7809785/
https://www.ncbi.nlm.nih.gov/pubmed/33448699
http://dx.doi.org/10.1002/prp2.699

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