Cargando…
Nitric Oxide and S-Nitrosylation in Cardiac Regulation: G Protein-Coupled Receptor Kinase-2 and β-Arrestins as Targets
Cardiac diseases including heart failure (HF), are the leading cause of morbidity and mortality globally. Among the prominent characteristics of HF is the loss of β-adrenoceptor (AR)-mediated inotropic reserve. This is primarily due to the derangements in myocardial regulatory signaling proteins, G...
Autores principales: | Kayki-Mutlu, Gizem, Koch, Walter J. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7825736/ https://www.ncbi.nlm.nih.gov/pubmed/33430208 http://dx.doi.org/10.3390/ijms22020521 |
Ejemplares similares
-
Sponging Away Cardiac Injury: A Natural Compound Shows Cardioprotective Promise
por: Kayki-Mutlu, Gizem, et al.
Publicado: (2022) -
Competing G protein-coupled receptor kinases balance G protein and β-arrestin signaling
por: Heitzler, Domitille, et al.
Publicado: (2012) -
G Protein–Coupled Receptor Kinase 2, With β-Arrestin 2, Impairs Insulin-Induced Akt/Endothelial Nitric Oxide Synthase Signaling in ob/ob Mouse Aorta
por: Taguchi, Kumiko, et al.
Publicado: (2012) -
Editorial: G Protein-Coupled Receptor Kinases (GRKs) and β-Arrestins: New Insights Into Disease Regulators
por: Hattori, Yuichi, et al.
Publicado: (2020) -
Regulation of β cell glucokinase by S-nitrosylation and association with nitric oxide synthase
por: Rizzo, Megan A., et al.
Publicado: (2003)