Cargando…

CCL2/MCP-1 signaling drives extracellular matrix turnover by diverse macrophage subsets()

Macrophage plasticity, cellular origin, and phenotypic heterogeneity are perpetual challenges for studies addressing the biology of this pivotal immune cell in development, homeostasis, and tissue remodeling/repair. Consequently, a myriad of macrophage subtypes has been described in these contexts....

Descripción completa

Detalles Bibliográficos
Autores principales:	Jürgensen, Henrik J., Silva, Lakmali M., Krigslund, Oliver, van Putten, Sander, Madsen, Daniel H., Behrendt, Niels, Engelholm, Lars H., Bugge, Thomas H.
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Elsevier 2019
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7852312/ https://www.ncbi.nlm.nih.gov/pubmed/33543002 http://dx.doi.org/10.1016/j.mbplus.2019.03.002

Ejemplares similares

Tumor cell MT1-MMP is dispensable for osteosarcoma tumor growth, bone degradation and lung metastasis
por: Ingvarsen, Signe Z., et al.
Publicado: (2020)

The Collagen Receptor uPARAP in Malignant Mesothelioma: A Potential Diagnostic Marker and Therapeutic Target
por: Çakılkaya, Pınar, et al.
Publicado: (2021)

The collagen receptor uPARAP/Endo180 in tissue degradation and cancer (Review)
por: MELANDER, MARIA C., et al.
Publicado: (2015)

Intracellular collagen degradation mediated by uPARAP/Endo180 is a major pathway of extracellular matrix turnover during malignancy
por: Curino, Alejandro C., et al.
Publicado: (2005)

Differential Actions of the Endocytic Collagen Receptor uPARAP/Endo180 and the Collagenase MMP-2 in Bone Homeostasis
por: Madsen, Daniel H., et al.
Publicado: (2013)

Immune regulation by fibroblasts in tissue injury depends on uPARAP-mediated uptake of collectins
por: Jürgensen, Henrik J., et al.
Publicado: (2019)

Uncovering mediators of collagen degradation in the tumor microenvironment
por: Thorseth, Marie-Louise, et al.
Publicado: (2022)

Osteosarcoma and Metastasis Associated Bone Degradation—A Tale of Osteoclast and Malignant Cell Cooperativity
por: Nørregaard, Kirstine Sandal, et al.
Publicado: (2021)

M2-like macrophages are responsible for collagen degradation through a mannose receptor–mediated pathway
por: Madsen, Daniel H., et al.
Publicado: (2013)

Monocyte Chemoattractant Protein-1 (MCP-1/CCL2) Drives Activation of Bone Remodelling and Skeletal Metastasis
por: Mulholland, Bridie S., et al.
Publicado: (2019)

CCL2/MCP-1 modulation of microglial activation and proliferation
por: Hinojosa, Ara E, et al.
Publicado: (2011)

Proteolytic processing and inactivation of CCL2/MCP-1 by meprins
por: Herzog, Christian, et al.
Publicado: (2016)

The collagen receptor uPARAP/Endo180 as a novel target for antibody-drug conjugate mediated treatment of mesenchymal and leukemic cancers
por: Nielsen, Christoffer Fagernæs, et al.
Publicado: (2017)

Detection of human MCP-4/CCL13 isoforms by SELDI immunoaffinity capture
por: Rossi, Leonardo, et al.
Publicado: (2006)

CCL2/MCP-I Genotype-Phenotype Relationship in Latent Tuberculosis Infection
por: Hussain, Rabia, et al.
Publicado: (2011)

Hypoxia-inducible factor-1 (HIF-1) is involved in the regulation of hypoxia-stimulated expression of monocyte chemoattractant protein-1 (MCP-1/CCL2) and MCP-5 (Ccl12) in astrocytes
por: Mojsilovic-Petrovic, Jelena, et al.
Publicado: (2007)

Mammalian Glycosylation Patterns Protect Citrullinated Chemokine MCP-1/CCL2 from Partial Degradation
por: Korchynskyi, Olexandr, et al.
Publicado: (2023)

Accelerated extracellular matrix turnover during exacerbations of COPD
por: Sand, Jannie M. B., et al.
Publicado: (2015)

Pro-Inflammatory Chemokine CCL2 (MCP-1) Promotes Healing in Diabetic Wounds by Restoring the Macrophage Response
por: Wood, Stephen, et al.
Publicado: (2014)

Serum levels of NLRC4 and MCP-2/CCL8 in patients with active Crohn’s disease
por: Irak, Kader, et al.
Publicado: (2021)

Imaging collagen degradation in vivo highlights a key role for M2-polarized macrophages in extracellular matrix degradation
por: Madsen, Daniel H, et al.
Publicado: (2013)

Inhibitory Monoclonal Antibodies against Mouse Proteases Raised in Gene-Deficient Mice Block Proteolytic Functions in vivo
por: Lund, Ida K., et al.
Publicado: (2012)

Live Imaging and Quantification of Neutrophil Extracellular Trap Formation
por: Silva, Lakmali Munasinghage, et al.
Publicado: (2021)

Induction of CCL8/MCP-2 by Mycobacteria through the Activation of TLR2/PI3K/Akt Signaling Pathway
por: Liu, Haipeng, et al.
Publicado: (2013)

Central Role for MCP-1/CCL2 in Injury-Induced Inflammation Revealed by In Vitro, In Silico, and Clinical Studies
por: Ziraldo, Cordelia, et al.
Publicado: (2013)

Extracellular Matrix Turnover: a Balance between MMPs and their Inhibitors
por: Yalcinkaya, Emre, et al.
Publicado: (2014)

Quantifying extracellular matrix turnover in human lung scaffold cultures
por: Rosmark, Oskar, et al.
Publicado: (2018)

Association of Early Nasopharyngeal Immune Markers With COVID-19 Clinical Outcome: Predictive Value of CCL2/MCP-1
por: Sierra, Beatriz, et al.
Publicado: (2020)

-2518A/G polymorphism of monocyte chemotactic protein 1 (MCP-1/CCL2) is associated with cutaneous mastocytosis
por: Ługowska-Umer, Hanna, et al.
Publicado: (2022)

Macrophage-Derived Factors with the Potential to Contribute to Pathogenicity of HIV-1 and HIV-2: Role of CCL-2/MCP-1
por: Gao, Chunling, et al.
Publicado: (2023)

Pivotal Role of Matrix Metalloproteinase 13 in Extracellular Matrix Turnover in Idiopathic Pulmonary Fibrosis
por: Nkyimbeng, Takwi, et al.
Publicado: (2013)

The source of matrix-degrading enzymes in human cancer: Problems of research reproducibility and possible solutions
por: Madsen, Daniel H., et al.
Publicado: (2015)

Low-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model
por: Gaiad, Thaís P., et al.
Publicado: (2017)

Extracellular matrix turnover, angiogenesis and endothelial function in acute lung injury: relationship to pulmonary dysfunction and outcome
por: Sayed, S, et al.
Publicado: (2012)

Targeting the pro-inflammatory factor CCL2 (MCP-1) with Bindarit for influenza A (H7N9) treatment
por: Wolf, Stefan, et al.
Publicado: (2017)

Biomarkers of extracellular matrix turnover are associated with emphysema and eosinophilic-bronchitis in COPD
por: Bihlet, Asger Reinstrup, et al.
Publicado: (2017)

Turnover of the extracellular polymeric matrix of granules performing biological phosphate removal
por: Tomás-Martínez, Sergio, et al.
Publicado: (2023)

Biochemical Analysis of Matrix Metalloproteinase Activation of Chemokines CCL15 and CCL23 and Increased Glycosaminoglycan Binding of CCL16
por: Starr, Amanda E., et al.
Publicado: (2012)

MCP-1 is overexpressed in triple-negative breast cancers and drives cancer invasiveness and metastasis
por: Dutta, Pranabananda, et al.
Publicado: (2018)

Expression of the inflammatory chemokines CCL5, CCL3 and CXCL10 in juvenile idiopathic arthritis, and demonstration of CCL5 production by an atypical subset of CD8+ T cells
por: Pharoah, Daniel S, et al.
Publicado: (2006)

Cannot write session to /tmp/vufind_sessions/sess_6l78f3kaj89d0nmd36eoe8fffa