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Sarcoplasmic reticular Ca(2+)-ATPase inhibition paradoxically upregulates murine skeletal muscle Na(v)1.4 function

Skeletal muscle Na(+) channels possess Ca(2+)- and calmodulin-binding sites implicated in Nav1.4 current (I(Na)) downregulation following ryanodine receptor (RyR1) activation produced by exchange protein directly activated by cyclic AMP or caffeine challenge, effects abrogated by the RyR1-antagonist...

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Detalles Bibliográficos
Autores principales:	Liu, Sean X., Matthews, Hugh R., Huang, Christopher L.-H.
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Nature Publishing Group UK 2021
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7854688/ https://www.ncbi.nlm.nih.gov/pubmed/33531589 http://dx.doi.org/10.1038/s41598-021-82493-w

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