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Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes

Detalles Bibliográficos
Autores principales: Janickova, Lucia, Rechberger, Karin Farah, Wey, Lucas, Schwaller, Beat
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7863247/
https://www.ncbi.nlm.nih.gov/pubmed/33546676
http://dx.doi.org/10.1186/s13229-020-00404-8
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author Janickova, Lucia
Rechberger, Karin Farah
Wey, Lucas
Schwaller, Beat
author_facet Janickova, Lucia
Rechberger, Karin Farah
Wey, Lucas
Schwaller, Beat
author_sort Janickova, Lucia
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spelling pubmed-78632472021-02-05 Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes Janickova, Lucia Rechberger, Karin Farah Wey, Lucas Schwaller, Beat Mol Autism Correction BioMed Central 2021-02-05 /pmc/articles/PMC7863247/ /pubmed/33546676 http://dx.doi.org/10.1186/s13229-020-00404-8 Text en © The Author(s) 2021 Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.
spellingShingle Correction
Janickova, Lucia
Rechberger, Karin Farah
Wey, Lucas
Schwaller, Beat
Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title_full Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title_fullStr Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title_full_unstemmed Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title_short Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
title_sort correction to: absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory pvalb neurons in vivo: a point of convergence of autism spectrum disorder (asd) risk gene phenotypes
topic Correction
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7863247/
https://www.ncbi.nlm.nih.gov/pubmed/33546676
http://dx.doi.org/10.1186/s13229-020-00404-8
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