Cargando…
Correction to: Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
Autores principales: | Janickova, Lucia, Rechberger, Karin Farah, Wey, Lucas, Schwaller, Beat |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7863247/ https://www.ncbi.nlm.nih.gov/pubmed/33546676 http://dx.doi.org/10.1186/s13229-020-00404-8 |
Ejemplares similares
-
Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes
por: Janickova, Lucia, et al.
Publicado: (2020) -
Parvalbumin-Deficiency Accelerates the Age-Dependent ROS Production in Pvalb Neurons in vivo: Link to Neurodevelopmental Disorders
por: Janickova, Lucia, et al.
Publicado: (2020) -
17-β estradiol increases parvalbumin levels in Pvalb heterozygous mice and attenuates behavioral phenotypes with relevance to autism core symptoms
por: Filice, Federica, et al.
Publicado: (2018) -
The Parvalbumin Hypothesis of Autism Spectrum Disorder
por: Filice, Federica, et al.
Publicado: (2020) -
Profiling parvalbumin interneurons using iPSC: challenges and perspectives for Autism Spectrum Disorder (ASD)
por: Filice, Federica, et al.
Publicado: (2020)