NADPH and Mito-Apocynin Treatment Protects Against KA-Induced Excitotoxic Injury Through Autophagy Pathway

AIM: Previous research recognizes that NADPH can produce reduced glutathione (GSH) as a coenzyme and produce ROS as a substrate of NADPH oxidase (NOX). Besides, excessive activation of glutamate receptors results in mitochondrial impairment. The study aims at spelling out the effects of NADPH and Mi...

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Detalles Bibliográficos
Autores principales: Liu, Na, Lin, Miao-Miao, Huang, Si-Si, Liu, Zi-Qi, Wu, Jun-Chao, Liang, Zhong-Qin, Qin, Zheng-Hong, Wang, Yan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Cell and Developmental Biology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7905037/
https://www.ncbi.nlm.nih.gov/pubmed/33644049
http://dx.doi.org/10.3389/fcell.2021.612554

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7905037/
https://www.ncbi.nlm.nih.gov/pubmed/33644049
http://dx.doi.org/10.3389/fcell.2021.612554

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