DNA- and telomere-damage does not limit lifespan: evidence from rapamycin

Failure of rapamycin to extend lifespan in DNA repair mutant and telomerase-knockout mice, while extending lifespan in normal mice, indicates that neither DNA damage nor telomere shortening limits normal lifespan or causes normal aging.

Detalles Bibliográficos
Autor principal: Blagosklonny, Mikhail V.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals 2021
Materias:
Research Perspective
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7906135/
https://www.ncbi.nlm.nih.gov/pubmed/33578394
http://dx.doi.org/10.18632/aging.202674

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7906135/
https://www.ncbi.nlm.nih.gov/pubmed/33578394
http://dx.doi.org/10.18632/aging.202674

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