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DNA- and telomere-damage does not limit lifespan: evidence from rapamycin
Failure of rapamycin to extend lifespan in DNA repair mutant and telomerase-knockout mice, while extending lifespan in normal mice, indicates that neither DNA damage nor telomere shortening limits normal lifespan or causes normal aging.
Autor principal: | Blagosklonny, Mikhail V. |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7906135/ https://www.ncbi.nlm.nih.gov/pubmed/33578394 http://dx.doi.org/10.18632/aging.202674 |
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