Cargando…
NFAT5 directs hyperosmotic stress-induced fibrin deposition and macrophage infiltration via PAI-1 in endothelium
Although stress can significantly promote atherosclerosis, the underlying mechanisms are still not completely understood. Here we successfully unveiled that high salt-induced nuclear factor of activated T cells 5 (NFAT5) control the endothelial-dependent fibrinolytic activity and the inflammatory ad...
Autores principales: | Ma, Pingping, Li, Guang, Jiang, Xiaorui, Shen, Xinkun, Li, Hong, Yang, Li, Liu, Wanqian |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7906158/ https://www.ncbi.nlm.nih.gov/pubmed/33410782 http://dx.doi.org/10.18632/aging.202330 |
Ejemplares similares
-
NFAT5 mediates hypertonic stress-induced atherosclerosis via activating NLRP3 inflammasome in endothelium
por: Ma, Pingping, et al.
Publicado: (2019) -
NFAT5 promotes oral squamous cell carcinoma progression in a hyperosmotic environment
por: Yoshimoto, Shohei, et al.
Publicado: (2020) -
Regulation of the hyperosmotic induction of aquaporin 5 and VEGF in retinal pigment epithelial cells: Involvement of NFAT5
por: Hollborn, Margrit, et al.
Publicado: (2015) -
Age-Related Increase of Collagen/Fibrin Deposition and High PAI-1 Production in Human Nasal Polyps
por: Jo, Ara, et al.
Publicado: (2022) -
Nfat5 is involved in the hyperosmotic regulation of Tmem184b: a putative modulator of ibuprofen transport in renal MDCK I cells
por: Rasmussen, Rune Nørgaard, et al.
Publicado: (2019)