Cargando…
Mechanisms of Oxidative Stress and Therapeutic Targets following Intracerebral Hemorrhage
Oxidative stress (OS) is induced by the accumulation of reactive oxygen species (ROS) following intracerebral hemorrhage (ICH) and plays an important role in secondary brain injury caused by the inflammatory response, apoptosis, autophagy, and blood-brain barrier (BBB) disruption. This review summar...
Autores principales: | Yao, Zhenjia, Bai, Qinqin, Wang, Gaiqing |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Hindawi
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7920740/ https://www.ncbi.nlm.nih.gov/pubmed/33688394 http://dx.doi.org/10.1155/2021/8815441 |
Ejemplares similares
-
Neuroprotection by Nrf2 via modulating microglial phenotype and phagocytosis after intracerebral hemorrhage
por: Liang, Chuntian, et al.
Publicado: (2023) -
Ferroptosis, a Regulated Neuronal Cell Death Type After Intracerebral Hemorrhage
por: Bai, Qinqin, et al.
Publicado: (2020) -
Oxidative Stress in Intracerebral Hemorrhage: Sources, Mechanisms, and Therapeutic Targets
por: Hu, Xin, et al.
Publicado: (2016) -
Long-term outcomes of monascin – a novel dual peroxisome
proliferator-activated receptor γ/nuclear factor-erythroid 2 related factor-2
agonist in experimental intracerebral hemorrhage
por: Fu, Pengcheng, et al.
Publicado: (2020) -
Microglia: A Double-Edged Sword in Intracerebral Hemorrhage From Basic Mechanisms to Clinical Research
por: Liu, Jiachen, et al.
Publicado: (2021)