Cargando…
Myocardial Hypertrophy and Compensatory Increase in Systolic Function in a Mouse Model of Oxidative Stress
Free radicals, or reactive oxygen species, have been implicated as one of the primary causes of myocardial pathologies elicited by chronic diseases and age. The imbalance between pro-oxidants and antioxidants, termed “oxidative stress”, involves several pathological changes in mouse hearts, includin...
Autores principales: | Varshney, Rohan, Ranjit, Rojina, Chiao, Ying Ann, Kinter, Michael, Ahn, Bumsoo |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7921997/ https://www.ncbi.nlm.nih.gov/pubmed/33670798 http://dx.doi.org/10.3390/ijms22042039 |
Ejemplares similares
-
Cancer cachexia in a mouse model of oxidative stress
por: Brown, Jacob L., et al.
Publicado: (2020) -
Using MRI to measure in vivo free radical production and perfusion dynamics in a mouse model of elevated oxidative stress and neurogenic atrophy
por: Ahn, Bumsoo, et al.
Publicado: (2019) -
Acylated Ghrelin Receptor Agonist HM01 Decreases Lean Body and Muscle Mass, but Unacylated Ghrelin Protects against Redox-Dependent Sarcopenia
por: Ranjit, Rojina, et al.
Publicado: (2022) -
COMPENSATORY HYPERTROPHY OF THE SPLEEN
por: MacKay, Eaton M., et al.
Publicado: (1931) -
Mitochondrial oxidative stress impairs contractile function but paradoxically increases muscle mass via fibre branching
por: Ahn, Bumsoo, et al.
Publicado: (2019)