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Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells

Chronic liver diseases caused by various pathogenesis are marked by inflammatory infiltration and wound healing reaction, while their normal regeneration ability is impaired. The unbalance between the generation and the degradation of extracellular matrix (ECM) leads to collagen accumulation and dev...

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Autores principales: Shu, Yongxiang, Liu, Xuyou, Huang, Haifeng, Wen, Qi, Shu, Jianchang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Netherlands 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7925445/
https://www.ncbi.nlm.nih.gov/pubmed/33609264
http://dx.doi.org/10.1007/s11033-021-06171-w
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author Shu, Yongxiang
Liu, Xuyou
Huang, Haifeng
Wen, Qi
Shu, Jianchang
author_facet Shu, Yongxiang
Liu, Xuyou
Huang, Haifeng
Wen, Qi
Shu, Jianchang
author_sort Shu, Yongxiang
collection PubMed
description Chronic liver diseases caused by various pathogenesis are marked by inflammatory infiltration and wound healing reaction, while their normal regeneration ability is impaired. The unbalance between the generation and the degradation of extracellular matrix (ECM) leads to collagen accumulation and develops into liver fibrosis. Inflammation, oxidative stress, and autophagy interact closely in the pathogenesis of hepatic fibrosis. Reactive Oxygen Species (ROS) can not only stimulate Kupffer cells to release massive inflammatory factors, but induce autophagy. However, the latter may suppress inflammatory reaction by inhibiting proinflammatory complex formation directly, and removing damaged organelles or pathogenic microorganism indirectly. At present, effective anti-fibrosis drugs are still lacking. Previous studies have found various natural compounds enabled liver protection through anti-inflammatory, antioxidant, and other mechanisms. In recent years, autophagy, a vital life activity, has been found to be involved in the mechanism of liver fibrosis. As a new target, developing anti-liver fibrosis drugs that regulate the activity of autophagy is very promising. In this review, we summarize the latest studies about natural compounds in the treatment of liver fibrosis by regulating autophagy.
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spelling pubmed-79254452021-03-19 Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells Shu, Yongxiang Liu, Xuyou Huang, Haifeng Wen, Qi Shu, Jianchang Mol Biol Rep Mini Review Article Chronic liver diseases caused by various pathogenesis are marked by inflammatory infiltration and wound healing reaction, while their normal regeneration ability is impaired. The unbalance between the generation and the degradation of extracellular matrix (ECM) leads to collagen accumulation and develops into liver fibrosis. Inflammation, oxidative stress, and autophagy interact closely in the pathogenesis of hepatic fibrosis. Reactive Oxygen Species (ROS) can not only stimulate Kupffer cells to release massive inflammatory factors, but induce autophagy. However, the latter may suppress inflammatory reaction by inhibiting proinflammatory complex formation directly, and removing damaged organelles or pathogenic microorganism indirectly. At present, effective anti-fibrosis drugs are still lacking. Previous studies have found various natural compounds enabled liver protection through anti-inflammatory, antioxidant, and other mechanisms. In recent years, autophagy, a vital life activity, has been found to be involved in the mechanism of liver fibrosis. As a new target, developing anti-liver fibrosis drugs that regulate the activity of autophagy is very promising. In this review, we summarize the latest studies about natural compounds in the treatment of liver fibrosis by regulating autophagy. Springer Netherlands 2021-02-20 2021 /pmc/articles/PMC7925445/ /pubmed/33609264 http://dx.doi.org/10.1007/s11033-021-06171-w Text en © The Author(s) 2021 Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.
spellingShingle Mini Review Article
Shu, Yongxiang
Liu, Xuyou
Huang, Haifeng
Wen, Qi
Shu, Jianchang
Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title_full Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title_fullStr Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title_full_unstemmed Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title_short Research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
title_sort research progress of natural compounds in anti-liver fibrosis by affecting autophagy of hepatic stellate cells
topic Mini Review Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7925445/
https://www.ncbi.nlm.nih.gov/pubmed/33609264
http://dx.doi.org/10.1007/s11033-021-06171-w
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