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Likelihood of amyloid formation in COVID-19-induced ARDS
Severe coronavirus disease 2019 (COVID-19) infection leads to multifactorial acute respiratory distress syndrome (ARDS), with little therapeutic success. The pathophysiology associated with ARDS or post-ARDS is not yet well understood. We hypothesize that amyloid formation occurring due to protein h...
Autores principales: | , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier Ltd.
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8007089/ https://www.ncbi.nlm.nih.gov/pubmed/33795156 http://dx.doi.org/10.1016/j.tim.2021.03.008 |
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author | Sinha, Nabodita Thakur, Ashwani K. |
author_facet | Sinha, Nabodita Thakur, Ashwani K. |
author_sort | Sinha, Nabodita |
collection | PubMed |
description | Severe coronavirus disease 2019 (COVID-19) infection leads to multifactorial acute respiratory distress syndrome (ARDS), with little therapeutic success. The pathophysiology associated with ARDS or post-ARDS is not yet well understood. We hypothesize that amyloid formation occurring due to protein homeostasis disruption can be one of the complications associated with COVID-19-induced-ARDS. |
format | Online Article Text |
id | pubmed-8007089 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Elsevier Ltd. |
record_format | MEDLINE/PubMed |
spelling | pubmed-80070892021-03-30 Likelihood of amyloid formation in COVID-19-induced ARDS Sinha, Nabodita Thakur, Ashwani K. Trends Microbiol Forum Severe coronavirus disease 2019 (COVID-19) infection leads to multifactorial acute respiratory distress syndrome (ARDS), with little therapeutic success. The pathophysiology associated with ARDS or post-ARDS is not yet well understood. We hypothesize that amyloid formation occurring due to protein homeostasis disruption can be one of the complications associated with COVID-19-induced-ARDS. Elsevier Ltd. 2021-11 2021-03-29 /pmc/articles/PMC8007089/ /pubmed/33795156 http://dx.doi.org/10.1016/j.tim.2021.03.008 Text en © 2021 Elsevier Ltd. All rights reserved. Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active. |
spellingShingle | Forum Sinha, Nabodita Thakur, Ashwani K. Likelihood of amyloid formation in COVID-19-induced ARDS |
title | Likelihood of amyloid formation in COVID-19-induced ARDS |
title_full | Likelihood of amyloid formation in COVID-19-induced ARDS |
title_fullStr | Likelihood of amyloid formation in COVID-19-induced ARDS |
title_full_unstemmed | Likelihood of amyloid formation in COVID-19-induced ARDS |
title_short | Likelihood of amyloid formation in COVID-19-induced ARDS |
title_sort | likelihood of amyloid formation in covid-19-induced ards |
topic | Forum |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8007089/ https://www.ncbi.nlm.nih.gov/pubmed/33795156 http://dx.doi.org/10.1016/j.tim.2021.03.008 |
work_keys_str_mv | AT sinhanabodita likelihoodofamyloidformationincovid19inducedards AT thakurashwanik likelihoodofamyloidformationincovid19inducedards |