Cargando…
Transforming growth factor-β1-induced N-cadherin drives cell–cell communication through connexin43 in osteoblast lineage
Gap junction (GJ) has been indicated to have an intimate correlation with adhesion junction. However, the direct interaction between them partially remains elusive. In the current study, we aimed to elucidate the role of N-cadherin, one of the core components in adhesion junction, in mediating conne...
Autores principales: | Yang, Yueyi, Liu, Wenjing, Wei, JieYa, Cui, Yujia, Zhang, Demao, Xie, Jing |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8044142/ https://www.ncbi.nlm.nih.gov/pubmed/33850101 http://dx.doi.org/10.1038/s41368-021-00119-3 |
Ejemplares similares
-
TGF-β1 facilitates cell–cell communication in osteocytes via connexin43- and pannexin1-dependent gap junctions
por: Liu, Wenjing, et al.
Publicado: (2019) -
Osteoblasts induce glucose-derived ATP perturbations in chondrocytes through noncontact communication: Osteoblasts induce glucose-derived ATP perturbations in chondrocytes
por: Wei, Jieya, et al.
Publicado: (2022) -
Premature Osteoblast Clustering by Enamel Matrix Proteins Induces Osteoblast Differentiation through Up-Regulation of Connexin 43 and N-Cadherin
por: Miron, Richard J., et al.
Publicado: (2011) -
Osteoblasts impair cholesterol synthesis in chondrocytes via Notch1 signalling
por: Yang, Yueyi, et al.
Publicado: (2021) -
Connexin 32 and connexin 43 are involved in lineage restriction of hepatic progenitor cells to hepatocytes
por: Pei, Haiyun, et al.
Publicado: (2017)