Cargando…

Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration

Detalles Bibliográficos
Autores principales: Sepehr, Afrah, Taheri, Fereshteh, Kandezi, Niyoosha, Motaghinejad, Majid, Safari, Sepideh, Mohammadi, Nilofar
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Wolters Kluwer - Medknow 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8106283/
https://www.ncbi.nlm.nih.gov/pubmed/34084314
http://dx.doi.org/10.4103/ijpvm.IJPVM_249_20
_version_ 1783689754190020608
author Sepehr, Afrah
Taheri, Fereshteh
Kandezi, Niyoosha
Motaghinejad, Majid
Safari, Sepideh
Mohammadi, Nilofar
author_facet Sepehr, Afrah
Taheri, Fereshteh
Kandezi, Niyoosha
Motaghinejad, Majid
Safari, Sepideh
Mohammadi, Nilofar
author_sort Sepehr, Afrah
collection PubMed
description
format Online
Article
Text
id pubmed-8106283
institution National Center for Biotechnology Information
language English
publishDate 2021
publisher Wolters Kluwer - Medknow
record_format MEDLINE/PubMed
spelling pubmed-81062832021-06-02 Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration Sepehr, Afrah Taheri, Fereshteh Kandezi, Niyoosha Motaghinejad, Majid Safari, Sepideh Mohammadi, Nilofar Int J Prev Med Editorial Wolters Kluwer - Medknow 2021-02-24 /pmc/articles/PMC8106283/ /pubmed/34084314 http://dx.doi.org/10.4103/ijpvm.IJPVM_249_20 Text en Copyright: © 2021 International Journal of Preventive Medicine https://creativecommons.org/licenses/by-nc-sa/4.0/This is an open access journal, and articles are distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as appropriate credit is given and the new creations are licensed under the identical terms.
spellingShingle Editorial
Sepehr, Afrah
Taheri, Fereshteh
Kandezi, Niyoosha
Motaghinejad, Majid
Safari, Sepideh
Mohammadi, Nilofar
Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title_full Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title_fullStr Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title_full_unstemmed Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title_short Preventive Role of Cannabinoids Derivate against Methylphenidate-Induced Oxidative Stress and Inflammation: The Hypothetical Function of Keap1/Nrf2/ARE Signaling and Proposal of a Treatment Strategy for Neurodegeneration
title_sort preventive role of cannabinoids derivate against methylphenidate-induced oxidative stress and inflammation: the hypothetical function of keap1/nrf2/are signaling and proposal of a treatment strategy for neurodegeneration
topic Editorial
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8106283/
https://www.ncbi.nlm.nih.gov/pubmed/34084314
http://dx.doi.org/10.4103/ijpvm.IJPVM_249_20
work_keys_str_mv AT sepehrafrah preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration
AT taherifereshteh preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration
AT kandeziniyoosha preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration
AT motaghinejadmajid preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration
AT safarisepideh preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration
AT mohammadinilofar preventiveroleofcannabinoidsderivateagainstmethylphenidateinducedoxidativestressandinflammationthehypotheticalfunctionofkeap1nrf2aresignalingandproposalofatreatmentstrategyforneurodegeneration