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Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo
Regulated cell proliferation is an effector mechanism of regeneration, whilst dysregulated cell proliferation is a feature of cancer. We have previously identified microRNA (miRNA) that regulate successful and failed human liver regeneration. We hypothesized that these regulators may directly modify...
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8131690/ https://www.ncbi.nlm.nih.gov/pubmed/34006907 http://dx.doi.org/10.1038/s41598-021-90009-9 |
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author | Salehi, Siamak Tavabie, Oliver D. Villanueva, Augusto Watson, Julie Darling, David Quaglia, Alberto Farzaneh, Farzin Aluvihare, Varuna R. |
author_facet | Salehi, Siamak Tavabie, Oliver D. Villanueva, Augusto Watson, Julie Darling, David Quaglia, Alberto Farzaneh, Farzin Aluvihare, Varuna R. |
author_sort | Salehi, Siamak |
collection | PubMed |
description | Regulated cell proliferation is an effector mechanism of regeneration, whilst dysregulated cell proliferation is a feature of cancer. We have previously identified microRNA (miRNA) that regulate successful and failed human liver regeneration. We hypothesized that these regulators may directly modify tumor behavior. Here we show that inhibition of miRNAs -503 and -23a, alone or in combination, enhances tumor proliferation in hepatocyte and non-hepatocyte derived cancers in vitro, driving more aggressive tumor behavior in vivo. Inhibition of miRNA-152 caused induction of DNMT1, site-specific methylation with associated changes in gene expression and in vitro and in vivo growth inhibition. Enforced changes in expression of two miRNA recapitulating changes observed in failed regeneration led to complete growth inhibition of multi-lineage cancers in vivo. Our results indicate that regulation of regeneration and tumor aggressiveness are concordant and that miRNA-based inhibitors of regeneration may constitute a novel treatment strategy for human cancers. |
format | Online Article Text |
id | pubmed-8131690 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-81316902021-05-25 Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo Salehi, Siamak Tavabie, Oliver D. Villanueva, Augusto Watson, Julie Darling, David Quaglia, Alberto Farzaneh, Farzin Aluvihare, Varuna R. Sci Rep Article Regulated cell proliferation is an effector mechanism of regeneration, whilst dysregulated cell proliferation is a feature of cancer. We have previously identified microRNA (miRNA) that regulate successful and failed human liver regeneration. We hypothesized that these regulators may directly modify tumor behavior. Here we show that inhibition of miRNAs -503 and -23a, alone or in combination, enhances tumor proliferation in hepatocyte and non-hepatocyte derived cancers in vitro, driving more aggressive tumor behavior in vivo. Inhibition of miRNA-152 caused induction of DNMT1, site-specific methylation with associated changes in gene expression and in vitro and in vivo growth inhibition. Enforced changes in expression of two miRNA recapitulating changes observed in failed regeneration led to complete growth inhibition of multi-lineage cancers in vivo. Our results indicate that regulation of regeneration and tumor aggressiveness are concordant and that miRNA-based inhibitors of regeneration may constitute a novel treatment strategy for human cancers. Nature Publishing Group UK 2021-05-18 /pmc/articles/PMC8131690/ /pubmed/34006907 http://dx.doi.org/10.1038/s41598-021-90009-9 Text en © The Author(s) 2021 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Article Salehi, Siamak Tavabie, Oliver D. Villanueva, Augusto Watson, Julie Darling, David Quaglia, Alberto Farzaneh, Farzin Aluvihare, Varuna R. Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title | Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title_full | Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title_fullStr | Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title_full_unstemmed | Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title_short | Regeneration linked miRNA modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
title_sort | regeneration linked mirna modify tumor phenotype and can enforce multi-lineage growth arrest in vivo |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8131690/ https://www.ncbi.nlm.nih.gov/pubmed/34006907 http://dx.doi.org/10.1038/s41598-021-90009-9 |
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