Cargando…
Cellular Tropism of SARS-CoV-2 across Human Tissues and Age-related Expression of ACE2 and TMPRSS2 in Immune-inflammatory Stromal Cells
Recently, emerging evidence has indicated that COVID-19 represents a major threat to older populations, but the underlying mechanisms remain unclear. The pathogen causing COVID-19 is acute respiratory syndrome coronavirus 2 (SARS-CoV-2). SARS-CoV-2 infection depends on the key entry factors, angiote...
Autor principal: | Zheng, Ming |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
JKL International LLC
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8139212/ https://www.ncbi.nlm.nih.gov/pubmed/34094637 http://dx.doi.org/10.14336/AD.2021.0429 |
Ejemplares similares
-
Emerging roles of SARS-CoV-2 Spike-ACE2 in immune evasion and pathogenesis
por: Baldari, Cosima T., et al.
Publicado: (2023) -
Human mesenchymal stromal cells do not express ACE2 and TMPRSS2 and are not permissive to SARS‐CoV‐2 infection
por: Avanzini, Maria A., et al.
Publicado: (2021) -
SARS-CoV-2 cellular tropism
por: Cagno, Valeria
Publicado: (2020) -
ACE2 partially dictates the host range and tropism of SARS-CoV-2
por: Gao, Shan, et al.
Publicado: (2020) -
ACE2 and TMPRSS2 Potential Involvement in Genetic Susceptibility to
SARS-COV-2 in Cancer Patients
por: Ravaioli, Sara, et al.
Publicado: (2020)